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Increased cardiac expression of tissue inhibitor of metalloproteinase-1 and tissue inhibitor of metalloproteinase-2 is related to cardiac fibrosis and dysfunction in the chronic pressure-overloaded human heart (2005)

Abstract
Background - Alterations in the balance of matrix metalloproteinases ( MMPs) and their specific tissue inhibitors (TIMPs) are involved in left ventricular (LV) remodeling. Whether their expression is related to interstitial fibrosis or LV dysfunction in patients with chronic pressure overload - induced LV hypertrophy, however, is unknown.

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Publisher Lippincott williams & wilkins
Repository Lirias is a research document repository at KULeuven (Belgium)
Keywords metalloproteinases, remodeling, hypertrophy, hypertension, collagen, matrix gene-expression, aortic-valve disease, diastolic dysfunction, myocardial-infarction, failure, hypertrophy, rats, replacement, progression, mechanisms
Type Description (Metadata) only, IT, article
Language English
Relation Circulation vol:112 issue:8 pages:1136-1144