A. A. M. Wilde

Publication List Details

Period

1997 - 2007

Number

353

Co-Authors

Diurnal variation of ventricular repolarization in a large family with LQT3-Brugada syndrome characterized by nocturnal sudden death (2006)

Haaksma, J., Wilde, A.A.M.

BACKGROUND In patients with long-QT syndrome type 3 (LQT3), symptoms occur particularly at rest or during sleep. As to the underlying mechanism, excessive prolongation of the QT interval at slow...

Overlap syndrome of cardiac sodium channel disease in mice carrying the equivalent mutation of human SCN5A-1795insD (2006)

Remme, C.A., Verkerk, A.O., Nuyens, D., Brunschot, S. Van, Belterman, C.N.W., ...

Background - Patients carrying the cardiac sodium channel (SCN5A) mutation 1795insD show sudden nocturnal death and signs of multiple arrhythmia syndromes including bradycardia, conduction delay, QT...

Voltage-gated sodium channels: Action players with many faces (2006)

Koopmann, T.T., Bezzina, C.R., Wilde, A.A.M.

Voltage-gated sodium channels are responsible for the upstroke of the action potential and thereby play an important role in propagation of the electrical impulse in excitable tissues like muscle,...

Female predominance and transmission distortion in the long-QT syndrome (2006)

Imboden, M., Swan, H., Denjoy, I., Langen, I.M. Van, Latinen-Forsblom, P.J., Napolitano, C., ...

BACKGROUND: Congenital long-QT syndrome is a disorder resulting in ventricular arrhythmias and sudden death. The most common forms of the long-QT syndrome, types 1 and 2, are caused by mutations in...

Structural abnormalities of the inferoseptal left ventricular wall detected by cardiac magnetic resonance imaging in carriers of hypertrophic cardiomyopathy mutations (2006)

Germans, T., Wilde, A.A.M., Dijkmans, P.A., Chai, W., Kamp, O., Pinto, Y.M., ...

OBJECTIVES The purpose of this study was to evaluate whether structural left ventricular (LV) abnormalities can be observed in hypertrophic cardiomyopathy (HCM) mutation carriers who have not yet...