within the Noncollagenous Domain of �3(IV) Collagen: A Janus-Faced Antigen (2008)
Alport Alloantibodies, Goodpasture Autoantibodies, Xu-ping Wang, Agnes B. Fogo, Selene Colon, Giovanna Giannico, ...
Alport posttransplantation anti–glomerular basement membrane (GBM) nephritis is mediated by alloantibodies against the noncollagenous (NC1) domains of the �3�4�5(IV) collagen network, which...
Dr Agnes Fogo-Chronic Kidney Disease: Can We Stop the Epidemic? (2007)
Includes descriptive metadata provided by producer in MP4 file: "Discovery Lecture Series - Videos - Dr Agnes Fogo-Chronic Kidney Disease: Can We Stop the Epidemic?"
Dr Agnes Fogo-Chronic Kidney Disease: Can We Stop the Epidemic? (2007)
Includes descriptive metadata provided by producer in MP4 file: "Discovery Lecture Series - Videos - Dr Agnes Fogo-Chronic Kidney Disease: Can We Stop the Epidemic?"
Plasminogen activator inhibitor-1 modulates adipocyte differentiation (2006)
Kanjanabuch, Talerngsak, Mao, Su-Li, Hao, Chuan-Ming, Tang, Yi-Wei, ...
Increased plasminogen activator inhibitor-1 (PAI-1) is linked to obesity and insulin resistance. However, the functional role of PAI-1 in adipocytes is unknown. This study was designed to investigate...
Fogo, Agnes B., Kashgarian, Michael
Traducción de: Diagnostic atlas of renal pathology : a companion to Brenner & Rector's the kidney, 7th edition
Chronic allograft nephropathy: expression and localization of PAI-1 and PPAR-{gamma} (2005)
Revelo, Monica P., Federspiel, Charles, Helderman, Harold, Fogo, Agnes B.
Background. Chronic allograft nephropathy (CAN) is a major cause of loss of renal allografts. Mechanisms postulated to be involved include sequelae of rejection, warm ischaemia time, drug toxicity,...
Chronic allograft nephropathy: expression and localization of PAI-1 and PPAR-{gamma} (2005)
Revelo, Monica P., Federspiel, Charles, Helderman, Harold, Fogo, Agnes B.
Background. Chronic allograft nephropathy (CAN) is a major cause of loss of renal allografts. Mechanisms postulated to be involved include sequelae of rejection, warm ischaemia time, drug toxicity,...
Chronic allograft nephropathy: expression and localization of PAI-1 and PPAR-{gamma} (2005)
Revelo, Monica P., Federspiel, Charles, Helderman, Harold, Fogo, Agnes B.
Background. Chronic allograft nephropathy (CAN) is a major cause of loss of renal allografts. Mechanisms postulated to be involved include sequelae of rejection, warm ischaemia time, drug toxicity,...
Mrukowicz, Jacek Z., Wetzel, J. Denise, Goral, Mehmet I., Fogo, Agnes B., Wright, Peter F., Dermody, Terence S.
To better understand mechanisms of persistent rotavirus infections of cultured cells, we established independent, persistently infected cultures of MA104 cells, using rotavirus strain SA11. The...
Nishida, Masashi, Fujinaka, Hidehiko, Matsusaka, Taiji, Price, James, Kon, Valentina, Fogo, Agnes B., ...
We examined the in vivo function of the angiotensin II type 1 receptor (Agtr1) on macrophages in renal fibrosis. Fourteen days after the induction of unilateral ureteral obstruction (UUO), wild-type...
Renal fibrosis: not just PAI-1 in the sky
A delicate balance exists between ECM synthesis and degradation such that interruption of the corresponding pathways results in increased plasminogen activator inhibitor-1 (PAI-1), pathological...
Mrukowicz, Jacek Z., Wetzel, J. Denise, Goral, Mehmet I., Fogo, Agnes B., Wright, Peter F., Dermody, Terence S.
To better understand mechanisms of persistent rotavirus infections of cultured cells, we established independent, persistently infected cultures of MA104 cells, using rotavirus strain SA11. The...
Nishida, Masashi, Fujinaka, Hidehiko, Matsusaka, Taiji, Price, James, Kon, Valentina, Fogo, Agnes B., ...
We examined the in vivo function of the angiotensin II type 1 receptor (Agtr1) on macrophages in renal fibrosis. Fourteen days after the induction of unilateral ureteral obstruction (UUO), wild-type...
Renal fibrosis: not just PAI-1 in the sky
A delicate balance exists between ECM synthesis and degradation such that interruption of the corresponding pathways results in increased plasminogen activator inhibitor-1 (PAI-1), pathological...
Lack of Integrin α1β1 Leads to Severe Glomerulosclerosis after Glomerular Injury
Chen, Xiwu, Moeckel, Gilbert, Morrow, Jason D., Cosgrove, Dominic, Harris, Raymond C., Fogo, Agnes B., ...
Severity of fibrosis after injury is determined by the nature of the injury and host genetic susceptibility. Metabolism of collagen, the major component of fibrotic lesions, is, in part, regulated by...
Ma, Li-Jun, Yang, Haichun, Gaspert, Ariana, Carlesso, Gianluca, Barty, Melissa M., Davidson, Jeffrey M., ...
Transforming growth factor-β1 (TGF-β1) and the renin-angiotensin-aldosterone system are key mediators in kidney fibrosis. Integrin αvβ6, a heterodimeric matrix receptor expressed in epithelia,...
Mechanisms of progression of chronic kidney disease
Chronic kidney disease (CKD) occurs in all age groups, including children. Regardless of the underlying cause, CKD is characterized by progressive scarring that ultimately affects all structures of...