Alberto J. Kaumann

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

The low-affinity site of the β1-adrenoceptor and its relevance to cardiovascular pharmacology (2008)

Kaumann, Alberto J., Molenaar, Peter

β-Adrenoceptor blocking agents (β-blockers) that at low concentrations antagonize cardiostimulant effects of catecholamines, but at high concentrations also cause cardiostimulation, have been...

Appearance of a ventricular 5-HT4 receptor-mediated inotropic response to serotonin in heart failure (2005)

Qvigstad, Eirik, Brattelid, Trond, Sjaastad, Ivar, Andressen, Kjetil Wessel, Krobert, Kurt A., Birkeland, Jon Arne, ...

Background: Current pharmacological treatment of congestive heart failure (CHF) addresses changes in neurohumoral stimulation or cardiac responsiveness to such stimulation. Yet, undiscovered...

(-)-CGP 12177 increases contractile force and hastens relaxation of human myocardial preparations through a propranolol-resistant state of the beta1-adrenoceptor (2003)

Sarsero, Doreen, Russell, Fraser D., Lynham, James A., Rabnott, Glenn, Yang, Ian, Fong, Kwun M., ...

Two forms of the activated beta(1)-adrenoceptor exist, one that is stabilized by (-)-noradrenaline and is sensitive to blockade by (-)-propranolol and another which is stabilized by partial agonists...

(-)-CGP 12177 increases contractile force and hastens relaxation of human myocardial preparations through a propranolol-resistant state of the beta1-adrenoceptor (2003)

Sarsero, Doreen, Russell, Fraser D., Lynham, James A., Rabnott, Glenn, Yang, Ian, Fong, Kwun M., ...

Two forms of the activated beta(1)-adrenoceptor exist, one that is stabilized by (-)-noradrenaline and is sensitive to blockade by (-)-propranolol and another which is stabilized by partial agonists...

L-type calcium current and contractility in ventricular myocytes from mice overexpressing the cardiac {beta}2-adrenoceptor (1999)

Heubach, Jürgen F, Trebeß, Ina, Wettwer, Erich, Himmel, Herbert M, Michel, Martin C, Kaumann, Alberto J, ...

Objectives: The reported increase in basal activity of hearts from transgenic mice (TG4) overexpressing the human β2-adrenoceptor (β2-AR) was explained by spontaneously active β2-ARs that...

Herz-[beta]-Rezeptoren [Herz-beta-Rezeptoren]. (1983)

Kaumann, Alberto J.

Aus: Zeitschrift für Kardiologie. 72. 1983, S. 63 - 82

Effects of (−)-RO363 at human atrial β-adrenoceptor subtypes, the human cloned β3-adrenoceptor and rodent intestinal β3-adrenoceptors

Molenaar, Peter, Sarsero, Doreen, Arch, Jonathan R S, Kelly, John, Henson, Sian M, Kaumann, Alberto J

Chronic treatment of patients with β-blockers causes atrial inotropic hyperresponsiveness through β2-adrenoceptors, 5-HT4 receptors and H2-receptors but apparently not through β1-adrenoceptors...

Stimulation of cyclic AMP-dependent protein kinase in rat atria by (–;)-CGP 12177 through an atypical β-adrenoceptor

Kaumann, Alberto J, Lynham, James A

Mammalian hearts possess an atypical β-adrenoceptor (non-β1, non-β2, non-β3) through which (–)-4-(3-t-butylamino-2-hydroxypropoxy)benzimidazol-2-one ((–)-CGP 12177) causes cardiostimulant...

Validity of (−)-[3H]-CGP 12177A as a radioligand for the ‘putative β4-adrenoceptor' in rat atrium

Sarsero, Doreen, Molenaar, Peter, Kaumann, Alberto J

We have recently suggested the existence in the heart of a ‘putative β4-adrenoceptor' based on the cardiostimulant effects of non-conventional partial agonists, compounds that cause...

Action potential shortening through the putative β4-adrenoceptor in ferret ventricle: comparison with β1- and β2-adrenoceptor-mediated effects

Lowe, Martin D, Grace, Andrew A, Vandenberg, Jamie I, Kaumann, Alberto J

The electrophysiological responses to (−)-CGP 12177 ((−)-4-(3-tertiarybutylamino-2-hydroxypropoxy) benzimidazol-2-one), an agonist for the putative β4-adrenoceptor, were investigated on isolated...

Putative β4-adrenoceptors in rat ventricle mediate increases in contractile force and cell Ca2+: comparison with atrial receptors and relationship to (−)-[3H]-CGP 12177 binding

Sarsero, Doreen, Molenaar, Peter, Kaumann, Alberto J, Freestone, Nicholas S

We identified putative β4-adrenoceptors by radioligand binding, measured increases in ventricular contractile force by (−)-CGP 12177 and (±)-cyanopindolol and demonstrated increased Ca2+...

Comparison of the affinity of β-blockers for two states of the β1-adrenoceptor in ferret ventricular myocardium

Lowe, Martin D, Lynham, James A, Grace, Andrew A, Kaumann, Alberto J

We compared the potency of 11 clinically available β-blockers as antagonists of the positive inotropic effects of (−)-isoprenaline and CGP12177 on ferret ventricular myocardium.(−)-CGP12177,...

Physiological antagonism between ventricular β1-adrenoceptors and α1-adrenoceptors but no evidence for β2- and β3-adrenoceptor function in murine heart

Heubach, Jürgen F, Rau, Thomas, Eschenhagen, Thomas, Ravens, Ursula, Kaumann, Alberto J

Murine left atrium lacks inotropic β2-adrenoceptor function. We investigated whether β2-adrenoceptors are involved in the cardiostimulant effects of (−)-adrenaline on spontaneously beating right...

Fading of 5-HT4 receptor-mediated inotropic responses to 5-hydroxytryptamine is caused by phosphodiesterase activity in porcine atrium

Kaumann, Alberto J, Levy, Finn Olav

Inotropic responses to 5-hydroxytryptamine (5-HT) in human and porcine atrium can fade, suggesting 5-HT4 receptor desensitization. De Maeyer et al., however, show in this issue that inhibition of...

Phosphodiesterase PDE3 blunts the positive inotropic and cyclic AMP enhancing effects of CGP12177 but not of noradrenaline in rat ventricle

Vargas, Maria Luisa, Hernandez, Jesus, Kaumann, Alberto J

The cardiostimulant effects of CGP12177, mediated through a β1-adrenoceptor site with low affinity for (−)-propranolol, are potentiated by the nonselective PDE inhibitor IBMX but the role of PDE...

BIBN4096BS is a potent competitive antagonist of the relaxant effects of α-CGRP on human temporal artery: comparison with CGRP(8-37)

Verheggen, Raphaela, Bumann, Katja, Kaumann, Alberto J

Release of CGRP during migraine may produce harmful dilatation of cranial arteries, thereby possibly causing pain. We have compared the antagonism by BIBN4096BS and CGRP(8-37) of the relaxant effects...

Octimibate, a potent non-prostanoid inhibitor of platelet aggregation, acts via the prostacyclin receptor

Merritt, Janet E., Hallam, Trevor J., Brown, Anthony M., Boyfield, Isobel, Cooper, David G., Hickey, Deirdre M.B., ...

1 Octimibate, 8-[(1,4,5-triphenyl-1H-imidazol-2-yl)oxy]octanoic acid, is reported to have antihrombotic properties, This is in addition to its antihyperlipidaemic effects which are due to inhibition...

Primate vascular responses to octimibate, a non-prostanoid agonist at the prostacyclin receptor

Merritt, Janet E., Brown, Anthony M., Bund, Stuart, Cooper, David G., Egan, John W., Hallam, Trevor J., ...

1 Octimibate is a potent inhibitor of human platelet aggregation, and appears to act (at least in part) through the prostacyclin receptor, as described in the preceding paper. Here, the vascular...