Basil Rigas

Chemopreventive agents induce oxidative stress in cancer cells leading to COX-2 overexpression and COX-2-independent cell death (2009)

Sun, Yu, Chen, Jie, Rigas, Basil

Chemopreventive agents generate oxidative stress, which culminates in cell death and may be part of a general mechanism of chemoprevention. The redox-responsive cyclooxygenase (COX)-2, overexpressed...

NO-Donating NSAIDs, PPARδ, and Cancer: Does PPARδ Contribute to Colon Carcinogenesis? (2008)

Gerardo G. Mackenzie, Shaheen Rasheed, William Wertheim, Basil Rigas

The chemopreventive NO-donating NSAIDs (NO-NSAIDs; NSAIDs with an NO-releasing moiety) modulate PPARδ and offer the opportunity to revisit the controversial role of PPARδ in...

NO-Donating NSAIDs, PPARδ, and Cancer: Does PPARδ Contribute to Colon Carcinogenesis? (2008)

Gerardo G. Mackenzie, Shaheen Rasheed, William Wertheim, Basil Rigas

The chemopreventive NO-donating NSAIDs (NO-NSAIDs; NSAIDs with an NO-releasing moiety) modulate PPARδ and offer the opportunity to revisit the controversial role of PPARδ in carcinogenesis (several...

NO-donating aspirin inhibits angiogenesis by suppressing VEGF expression in HT-29 human colon cancer mouse xenografts (2008)

Ouyang, Nengtai, Williams, Jennie L., Rigas, Basil

The inhibitory effect of NO-donating aspirin (NO-ASA) on colon cancer has been demonstrated in vivo and in vitro but its mechanism is still obscure. We investigated the effect of NO-ASA on...

NO-donating aspirin inhibits the activation of NF-{kappa}B in human cancer cell lines and Min mice (2008)

Williams, Jennie L., Ji, Ping, Ouyang, Nengtai, Liu, Xiaoping, Rigas, Basil

Nitric oxide-donating aspirin (NO-ASA) is a promising agent for the control of cancer, whose mechanism of action remains unclear. NF-κB is an important signaling molecule in the pathogenesis of...

NO-donating aspirin induces phase II enzymes in vitro and in vivo (2006)

Gao, Jianjun, Kashfi, Khosrow, Liu, Xiaoping, Rigas, Basil

Modulation of drug metabolizing enzymes, leading to facilitated elimination of carcinogens represents a successful strategy for cancer chemoprevention. Nitric oxide-donating aspirin (NO-ASA) is a...

NO-donating aspirin isomers downregulate peroxisome proliferator-activated receptor (PPAR){delta} expression in APCmin/+ mice proportionally to their tumor inhibitory effect: implications for the role of PPAR{delta} in carcinogenesis (2005)

Ouyang, Nengtai, Williams, Jennie L., Rigas, Basil

Nitric oxide donating aspirin (NO-ASA), consisting of a traditional ASA to which a NO-releasing moiety is covalently attached, is a promising chemoprevention agent against colon cancer. Its mechanism...

NO-donating aspirin induces phase II enzymes in vitro and in vivo (2005)

Gao, Jianjun, Kashfi, Khosrow, Liu, Xiaoping, Rigas, Basil

Modulation of drug metabolizing enzymes, leading to facilitated elimination of carcinogens represents a successful strategy for cancer chemoprevention. Nitric oxide-donating aspirin (NO-ASA) is a...

Regional response leading to tumorigenesis after sulindac in small and large intestine of mice with Apc mutations (2003)

Yang, Kan, Fan, Kunhua, Kurihara, Naoto, Shinozaki, Hiroharu, Rigas, Basil, Augenlicht, Leonard, ...

Sulindac and other NSAIDs have been widely studied as potential chemopreventive agents for colon cancer. Short-term studies have shown adenomatous polyps to regress in patients with familial...

The effect of lithocholic acid on proliferation and apoptosis during the early stages of colon carcinogenesis: differential effect on apoptosis in the presence of a colon carcinogen (2000)

Kozoni, Vassiliki, Tsioulias, George, Shiff, Steven, Rigas, Basil

Lithocholic acid (LCA) is implicated in human and experimental animal carcinogenesis. Its effect on apoptosis and proliferation of the colonic epithelium was studied in a 1,2-dimethylhydrazine...

Nitric oxide-donating aspirin inhibits β-catenin/T cell factor (TCF) signaling in SW480 colon cancer cells by disrupting the nuclear β-catenin–TCF association

Nath, Niharika, Kashfi, Khosrow, Chen, Jie, Rigas, Basil

Dysregulation of the Wnt pathway and altered β-catenin expression are central early events in colorectal carcinogenesis. We studied the ortho, meta, and para (o-, m-, and p-) positional isomers of...

Nitric oxide-donating aspirin induces apoptosis in human colon cancer cells through induction of oxidative stress

Gao, Jianjun, Liu, Xiaoping, Rigas, Basil

Nitric oxide-donating aspirin (NO-ASA) is a promising chemoprevention agent against colon cancer and other cancers. It consists of traditional ASA to which a NO-releasing moiety is bound through a...

Nitric oxide-donating aspirin inhibits β-catenin/T cell factor (TCF) signaling in SW480 colon cancer cells by disrupting the nuclear β-catenin–TCF association

Nath, Niharika, Kashfi, Khosrow, Chen, Jie, Rigas, Basil

Dysregulation of the Wnt pathway and altered β-catenin expression are central early events in colorectal carcinogenesis. We studied the ortho, meta, and para (o-, m-, and p-) positional isomers of...

Nitric oxide-donating aspirin induces apoptosis in human colon cancer cells through induction of oxidative stress

Gao, Jianjun, Liu, Xiaoping, Rigas, Basil

Nitric oxide-donating aspirin (NO-ASA) is a promising chemoprevention agent against colon cancer and other cancers. It consists of traditional ASA to which a NO-releasing moiety is bound through a...

NO-Donating NSAIDs, PPARδ, and Cancer: Does PPARδ Contribute to Colon Carcinogenesis?

Mackenzie, Gerardo G., Rasheed, Shaheen, Wertheim, William, Rigas, Basil

The chemopreventive NO-donating NSAIDs (NO-NSAIDs; NSAIDs with an NO-releasing moiety) modulate PPARδ and offer the opportunity to revisit the controversial role of PPARδ in carcinogenesis (several...

NO-donating aspirin inhibits angiogenesis by suppressing VEGF expression in HT-29 human colon cancer mouse xenografts

Ouyang, Nengtai, Williams, Jennie L., Rigas, Basil

The inhibitory effect of NO-donating aspirin (NO-ASA) on colon cancer has been demonstrated in vivo and in vitro but its mechanism is still obscure. We investigated the effect of NO-ASA on...