Brian K. Coombes

A novel inhibitor of Chlamydophila pneumoniaeprotein kinase D (PknD) inhibits phosphorylation of CdsD and suppresses bacterial replication (2009)

Johnson, Dustin L, Stone, Chris B, Bulir, David C, Coombes, Brian K, Mahony, James B

Abstract Background We have shown previously that Chlamydophila pneumoniae contains a dual-specific Ser/Thr protein kinase that phosphorylates CdsD, a structural component of the type III secretion...

RpoE fine tunes expression of a subset of SsrB-regulated virulence factors in Salmonella entericaserovar Typhimurium (2009)

Osborne, Suzanne E, Coombes, Brian K

Abstract Background The survival of Salmonella enterica within the intracellular host niche requires highly co-ordinated expression of virulence effectors predominantly regulated by the SsrAB...

factors in Salmonella enterica serovar Typhimurium (2009)

Bmc Microbiology, Suzanne E Osborne, Brian K Coombes, Suzanne E Osborne, Brian K Coombes

Research article RpoE fine tunes expression of a subset of SsrB-regulated virulence

Oral infection of mice with Salmonella entericaserovar Typhimurium causes meningitis and infection of the brain (2007)

Wickham, Mark E, Brown, Nat F, Provias, John, Finlay, B Brett, Coombes, Brian K

Abstract Background Salmonella meningitis is a rare and serious infection of the central nervous system following acute Salmonella enterica sepsis. For this pathogen, no appropriate model has been...

Oral infection of mice with Salmonella enterica serovar Typhimurium causes meningitis and infection of the brain (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Provias, John, Finlay, B. Brett, Coombes, Brian K.

BACKGROUND: Salmonella meningitis is a rare and serious infection of the central nervous system following acute Salmonella enterica sepsis. For this pathogen, no appropriate model has been reported...

Oral infection of mice with Salmonella enterica serovar Typhimurium causes meningitis and infection of the brain (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Provias, John, Finlay, B. Brett, Coombes, Brian K.

BACKGROUND: Salmonella meningitis is a rare and serious infection of the central nervous system following acute Salmonella enterica sepsis. For this pathogen, no appropriate model has been reported...

Virulence is positively selected by transmission success between mammalian hosts (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Boyle, Erin C., Coombes, Brian K., Finlay, B. Brett

Virulence, defined as damage to the host, is a trait of pathogens that evolutionary theory suggests benefits the pathogen in the ''struggle for existence'' [1]. Pathogens employ virulence mechanisms...

SseL is a salmonella-specific translocated effector integrated into the SsrB-controlled salmonella pathogenicity island 2 type III secretion system (2007)

Coombes, Brian K., Lowden, Michael J., Bishop, Jenna L., Wickham, Mark E., Brown, Nathaniel Francis, Duong, Nancy, ...

Bacterial pathogens use horizontal gene transfer to acquire virulence factors that influence host colonization, alter virulence traits, and ultimately shape the outcome of disease following...

Virulence is positively selected by transmission success between mammalian hosts (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Boyle, Erin C., Coombes, Brian K., Finlay, B. Brett

Virulence, defined as damage to the host, is a trait of pathogens that evolutionary theory suggests benefits the pathogen in the ''struggle for existence'' [1]. Pathogens employ virulence mechanisms...

SseL is a salmonella-specific translocated effector integrated into the SsrB-controlled salmonella pathogenicity island 2 type III secretion system (2007)

Coombes, Brian K., Lowden, Michael J., Bishop, Jenna L., Wickham, Mark E., Brown, Nathaniel Francis, Duong, Nancy, ...

Bacterial pathogens use horizontal gene transfer to acquire virulence factors that influence host colonization, alter virulence traits, and ultimately shape the outcome of disease following...

Virulence is positively selected by transmission success between mammalian hosts (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Boyle, Erin C., Coombes, Brian K., Finlay, B. Brett

Virulence, defined as damage to the host, is a trait of pathogens that evolutionary theory suggests benefits the pathogen in the ''struggle for existence'' [1]. Pathogens employ virulence mechanisms...

SseL is a salmonella-specific translocated effector integrated into the SsrB-controlled salmonella pathogenicity island 2 type III secretion system (2007)

Coombes, Brian K., Lowden, Michael J., Bishop, Jenna L., Wickham, Mark E., Brown, Nathaniel Francis, Duong, Nancy, ...

Bacterial pathogens use horizontal gene transfer to acquire virulence factors that influence host colonization, alter virulence traits, and ultimately shape the outcome of disease following...

Oral infection of mice with Salmonella enterica serovar Typhimurium causes meningitis and infection of the brain (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Provias, John, Finlay, B. Brett, Coombes, Brian K.

BACKGROUND: Salmonella meningitis is a rare and serious infection of the central nervous system following acute Salmonella enterica sepsis. For this pathogen, no appropriate model has been reported...

Virulence is positively selected by transmission success between mammalian hosts (2007)

Wickham, Mark E., Brown, Nathaniel Francis, Boyle, Erin C., Coombes, Brian K., Finlay, B. Brett

Virulence, defined as damage to the host, is a trait of pathogens that evolutionary theory suggests benefits the pathogen in the ''struggle for existence'' [1]. Pathogens employ virulence mechanisms...

SseL is a salmonella-specific translocated effector integrated into the SsrB-controlled salmonella pathogenicity island 2 type III secretion system (2007)

Coombes, Brian K., Lowden, Michael J., Bishop, Jenna L., Wickham, Mark E., Brown, Nathaniel Francis, Duong, Nancy, ...

Bacterial pathogens use horizontal gene transfer to acquire virulence factors that influence host colonization, alter virulence traits, and ultimately shape the outcome of disease following...

Crossing the Line: Selection and Evolution of Virulence Traits (2006)

Nat F. Brown, Mark E. Wickham, Brian K. Coombes, B. Brett Finlay

The evolution of pathogens presents a paradox. Pathogenic species are often absolutely dependent on their host species for their propagation through evolutionary time, yet the pathogenic lifestyle...

Mutational analysis of Salmonella translocated effector members SifA and SopD2 reveals domains implicated in translocation, subcellular localization and function (2006)

Brown, Nathaniel Francis, Szeto, Jason, Jiang, Xiuju, Coombes, Brian K., Finlay, B. Brett, Brumell, John H.

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen causing disease in several hosts. These bacteria use two distinct type III secretion systems that inject effector...

Bacterial genetic determinants of non-O157 STEC outbreaks and hemolytic-uremic syndrome after infection (2006)

Wickham, Mark E., Lupp, Claudia, Mascarenhas, Mariola, Vazquez, Alejandra, Coombes, Brian K., Brown, Nathaniel Francis, ...

Although O157:H7 Shiga toxin–producing Escherichia coli (STEC) are the predominant cause of hemolytic-uremic syndrome (HUS) in the world, non-O157:H7 serotypes are a medically important cause of...

Mutational analysis of Salmonella translocated effector members SifA and SopD2 reveals domains implicated in translocation, subcellular localization and function (2006)

Brown, Nathaniel Francis, Szeto, Jason, Jiang, Xiuju, Coombes, Brian K., Finlay, B. Brett, Brumell, John H.

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen causing disease in several hosts. These bacteria use two distinct type III secretion systems that inject effector...

Bacterial genetic determinants of non-O157 STEC outbreaks and hemolytic-uremic syndrome after infection (2006)

Wickham, Mark E., Lupp, Claudia, Mascarenhas, Mariola, Vazquez, Alejandra, Coombes, Brian K., Brown, Nathaniel Francis, ...

Although O157:H7 Shiga toxin–producing Escherichia coli (STEC) are the predominant cause of hemolytic-uremic syndrome (HUS) in the world, non-O157:H7 serotypes are a medically important cause of...

Mutational analysis of Salmonella translocated effector members SifA and SopD2 reveals domains implicated in translocation, subcellular localization and function (2006)

Brown, Nathaniel Francis, Szeto, Jason, Jiang, Xiuju, Coombes, Brian K., Finlay, B. Brett, Brumell, John H.

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen causing disease in several hosts. These bacteria use two distinct type III secretion systems that inject effector...

Bacterial genetic determinants of non-O157 STEC outbreaks and hemolytic-uremic syndrome after infection (2006)

Wickham, Mark E., Lupp, Claudia, Mascarenhas, Mariola, Vazquez, Alejandra, Coombes, Brian K., Brown, Nathaniel Francis, ...

Although O157:H7 Shiga toxin–producing Escherichia coli (STEC) are the predominant cause of hemolytic-uremic syndrome (HUS) in the world, non-O157:H7 serotypes are a medically important cause of...

Mutational analysis of Salmonella translocated effector members SifA and SopD2 reveals domains implicated in translocation, subcellular localization and function (2006)

Brown, Nathaniel Francis, Szeto, Jason, Jiang, Xiuju, Coombes, Brian K., Finlay, B. Brett, Brumell, John H.

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen causing disease in several hosts. These bacteria use two distinct type III secretion systems that inject effector...

Bacterial genetic determinants of non-O157 STEC outbreaks and hemolytic-uremic syndrome after infection (2006)

Wickham, Mark E., Lupp, Claudia, Mascarenhas, Mariola, Vazquez, Alejandra, Coombes, Brian K., Brown, Nathaniel Francis, ...

Although O157:H7 Shiga toxin–producing Escherichia coli (STEC) are the predominant cause of hemolytic-uremic syndrome (HUS) in the world, non-O157:H7 serotypes are a medically important cause of...

Salmonella Pathogenicity Island 2 Is Expressed Prior to Penetrating the Intestine (2005)

Nat F. Brown, Bruce A. Vallance, Brian K. Coombes, Yanet Valdez, Bryan A. Coburn, B. Brett Finlay

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine (2005)

Brown, Nathaniel Francis, Vallance, Bruce A., Coombes, Brian K., Vandez, Yanet, Coburn, Bryan A., Finlay, B. Brett

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine (2005)

Brown, Nathaniel Francis, Vallance, Bruce A., Coombes, Brian K., Vandez, Yanet, Coburn, Bryan A., Finlay, B. Brett

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid (2005)

Coombes, Brian K., Lowden, Michael J., Wickham, Mark E., Brown, Nathaniel Francis, Finlay, B. Brett

Short RNA regulatory molecules, microRNAs, and short interfering RNAs participate in a range of developmental gene networks by base-pairing with their target sequences. Consistent with these...

Genetic and molecular analysis of GogB, a phage-encoded type III-secreted substrate in Salmonella enterica serovar typhimurium with autonomous expression from its associated phage (2005)

Coombes, Brian K., Wickham, Mark E., Brown, Nathaniel Francis, Lemire, Sebastien, Bossi, Lionello, Hsiao, William W. L., ...

Salmonella enterica serovar Typhimurium is lysogenized by several temperate bacteriophages that encode lysogenic conversion genes, which can act as virulence factors during infection and contribute...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid (2005)

Coombes, Brian K., Lowden, Michael J., Wickham, Mark E., Brown, Nathaniel Francis, Finlay, B. Brett

Short RNA regulatory molecules, microRNAs, and short interfering RNAs participate in a range of developmental gene networks by base-pairing with their target sequences. Consistent with these...

Genetic and molecular analysis of GogB, a phage-encoded type III-secreted substrate in Salmonella enterica serovar typhimurium with autonomous expression from its associated phage (2005)

Coombes, Brian K., Wickham, Mark E., Brown, Nathaniel Francis, Lemire, Sebastien, Bossi, Lionello, Hsiao, William W. L., ...

Salmonella enterica serovar Typhimurium is lysogenized by several temperate bacteriophages that encode lysogenic conversion genes, which can act as virulence factors during infection and contribute...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid (2005)

Coombes, Brian K., Lowden, Michael J., Wickham, Mark E., Brown, Nathaniel Francis, Finlay, B. Brett

Short RNA regulatory molecules, microRNAs, and short interfering RNAs participate in a range of developmental gene networks by base-pairing with their target sequences. Consistent with these...

Genetic and molecular analysis of GogB, a phage-encoded type III-secreted substrate in Salmonella enterica serovar typhimurium with autonomous expression from its associated phage (2005)

Coombes, Brian K., Wickham, Mark E., Brown, Nathaniel Francis, Lemire, Sebastien, Bossi, Lionello, Hsiao, William W. L., ...

Salmonella enterica serovar Typhimurium is lysogenized by several temperate bacteriophages that encode lysogenic conversion genes, which can act as virulence factors during infection and contribute...

Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine (2005)

Brown, Nathaniel Francis, Vallance, Bruce A., Coombes, Brian K., Vandez, Yanet, Coburn, Bryan A., Finlay, B. Brett

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid (2005)

Coombes, Brian K., Lowden, Michael J., Wickham, Mark E., Brown, Nathaniel Francis, Finlay, B. Brett

Short RNA regulatory molecules, microRNAs, and short interfering RNAs participate in a range of developmental gene networks by base-pairing with their target sequences. Consistent with these...

Genetic and molecular analysis of GogB, a phage-encoded type III-secreted substrate in Salmonella enterica serovar typhimurium with autonomous expression from its associated phage (2005)

Coombes, Brian K., Wickham, Mark E., Brown, Nathaniel Francis, Lemire, Sebastien, Bossi, Lionello, Hsiao, William W. L., ...

Salmonella enterica serovar Typhimurium is lysogenized by several temperate bacteriophages that encode lysogenic conversion genes, which can act as virulence factors during infection and contribute...

Expression and secretion of Salmonella pathogenicity island-2 virulence genes in response to acidification exhibit differential requirements of a functional type III secretion apparatus and SsaL (2004)

Coombes, Brian K., Brown, Nathaniel Francis, Vandez, Yanet, Brumell, John H., Finlay, B. Brett

Salmonella pathogenicity island (SPI)-2 is pivotal to the intracellular survival of Salmonella and for virulence in mammals. SPI-2 encodes virulence factors (called effectors) that are translocated...

Expression and secretion of Salmonella pathogenicity island-2 virulence genes in response to acidification exhibit differential requirements of a functional type III secretion apparatus and SsaL (2004)

Coombes, Brian K., Brown, Nathaniel Francis, Vandez, Yanet, Brumell, John H., Finlay, B. Brett

Salmonella pathogenicity island (SPI)-2 is pivotal to the intracellular survival of Salmonella and for virulence in mammals. SPI-2 encodes virulence factors(called effectors) that are translocated...

Expression and secretion of Salmonella pathogenicity island-2 virulence genes in response to acidification exhibit differential requirements of a functional type III secretion apparatus and SsaL (2004)

Coombes, Brian K., Brown, Nathaniel Francis, Vandez, Yanet, Brumell, John H., Finlay, B. Brett

Salmonella pathogenicity island (SPI)-2 is pivotal to the intracellular survival of Salmonella and for virulence in mammals. SPI-2 encodes virulence factors (called effectors) that are translocated...

Expression and secretion of Salmonella pathogenicity island-2 virulence genes in response to acidification exhibit differential requirements of a functional type III secretion apparatus and SsaL (2004)

Coombes, Brian K., Brown, Nathaniel Francis, Vandez, Yanet, Brumell, John H., Finlay, B. Brett

Salmonella pathogenicity island (SPI)-2 is pivotal to the intracellular survival of Salmonella and for virulence in mammals. SPI-2 encodes virulence factors (called effectors) that are translocated...

SseA is required for translocation of Salmonella pathogenicity island-2 effectors into host cells (2003)

Coombes, Brian K., Brown, Nathaniel Francis, Kujat-Choy, Sonya, Vallance, Bruce A., Finlay, B. Brett

The Salmonella pathogenicity island-2 (SPI2) is a virulence locus on the bacterial chromosome required for intracellular proliferation and systemic infection in mice. Cell culture models and a murine...

SseA is required for translocation of Salmonella pathogenicity island-2 effectors into host cells (2003)

Coombes, Brian K., Brown, Nathaniel Francis, Kujat-Choy, Sonya, Vallance, Bruce A., Finlay, B. Brett

The Salmonella pathogenicity island-2 (SPI2) is a virulence locus on the bacterial chromosome required for intracellular proliferation and systemic infection in mice. Cell culture models and a murine...

SseA is required for translocation of Salmonella pathogenicity island-2 effectors into host cells (2003)

Coombes, Brian K., Brown, Nathaniel Francis, Kujat-Choy, Sonya, Vallance, Bruce A., Finlay, B. Brett

The Salmonella pathogenicity island-2 (SPI2) is a virulence locus on the bacterial chromosome required for intracellular proliferation and systemic infection in mice. Cell culture models and a murine...

SseA is required for translocation of Salmonella pathogenicity island-2 effectors into host cells (2003)

Coombes, Brian K., Brown, Nathaniel Francis, Kujat-Choy, Sonya, Vallance, Bruce A., Finlay, B. Brett

The Salmonella pathogenicity island-2 (SPI2) is a virulence locus on the bacterial chromosome required for intracellular proliferation and systemic infection in mice. Cell culture models and a murine...

Chlamydia pneumoniae Infection of Human Endothelial Cells Induces Proliferation of Smooth Muscle Cells via an Endothelial Cell-Derived Soluble Factor(s)

Coombes, Brian K., Mahony, James B.

An association of Chlamydia pneumoniae with atherosclerosis and coronary heart disease has been determined epidemiologically and by the detection of C. pneumoniae organisms in atherosclerotic lesions...

cDNA Array Analysis of Altered Gene Expression in Human Endothelial Cells in Response to Chlamydia pneumoniae Infection

Coombes, Brian K., Mahony, James B.

Strong epidemiological and pathological evidence supports a role for Chlamydia pneumoniae infection in atherosclerosis and human coronary heart disease. Animal models have shown that C. pneumoniae...

Analysis of the Contribution of Salmonella Pathogenicity Islands 1 and 2 to Enteric Disease Progression Using a Novel Bovine Ileal Loop Model and a Murine Model of Infectious Enterocolitis

Coombes, Brian K., Coburn, Bryan A., Potter, Andrew A., Gomis, Susantha, Mirakhur, Kuldip, Li, Yuling, ...

We have developed a novel ileal loop model for use in calves to analyze the contribution of Salmonella enterica serovar Typhimurium type III secretion systems to disease processes in vivo. Our model...

Salmonella Pathogenicity Island 2 Is Expressed Prior to Penetrating the Intestine

Brown, Nat F, Vallance, Bruce A, Coombes, Brian K, Valdez, Yanet, Coburn, Bryan A, Finlay, B. Brett

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Crossing the Line: Selection and Evolution of Virulence Traits

Brown, Nat F, Wickham, Mark E, Coombes, Brian K, Finlay, B. Brett

The evolution of pathogens presents a paradox. Pathogenic species are often absolutely dependent on their host species for their propagation through evolutionary time, yet the pathogenic lifestyle...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid

Coombes, Brian K., Wickham, Mark E., Lowden, Michael J., Brown, Nat F., Finlay, B. Brett

Salmonella enterica relies on a type III secretion system encoded in Salmonella pathogenicity island-2 (SPI-2) to survive and replicate within macrophages at systemic sites during typhoid. SPI-2...

Chlamydia pneumoniae Infection of Human Endothelial Cells Induces Proliferation of Smooth Muscle Cells via an Endothelial Cell-Derived Soluble Factor(s)

Coombes, Brian K., Mahony, James B.

An association of Chlamydia pneumoniae with atherosclerosis and coronary heart disease has been determined epidemiologically and by the detection of C. pneumoniae organisms in atherosclerotic lesions...

cDNA Array Analysis of Altered Gene Expression in Human Endothelial Cells in Response to Chlamydia pneumoniae Infection

Coombes, Brian K., Mahony, James B.

Strong epidemiological and pathological evidence supports a role for Chlamydia pneumoniae infection in atherosclerosis and human coronary heart disease. Animal models have shown that C. pneumoniae...

Analysis of the Contribution of Salmonella Pathogenicity Islands 1 and 2 to Enteric Disease Progression Using a Novel Bovine Ileal Loop Model and a Murine Model of Infectious Enterocolitis

Coombes, Brian K., Coburn, Bryan A., Potter, Andrew A., Gomis, Susantha, Mirakhur, Kuldip, Li, Yuling, ...

We have developed a novel ileal loop model for use in calves to analyze the contribution of Salmonella enterica serovar Typhimurium type III secretion systems to disease processes in vivo. Our model...

Salmonella Pathogenicity Island 2 Is Expressed Prior to Penetrating the Intestine

Brown, Nat F, Vallance, Bruce A, Coombes, Brian K, Valdez, Yanet, Coburn, Bryan A, Finlay, B. Brett

Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that causes disease in mice that resembles human typhoid. Typhoid pathogenesis consists of distinct phases in the...

Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid

Coombes, Brian K., Wickham, Mark E., Lowden, Michael J., Brown, Nat F., Finlay, B. Brett

Salmonella enterica relies on a type III secretion system encoded in Salmonella pathogenicity island-2 (SPI-2) to survive and replicate within macrophages at systemic sites during typhoid. SPI-2...

Crossing the Line: Selection and Evolution of Virulence Traits

Brown, Nat F, Wickham, Mark E, Coombes, Brian K, Finlay, B. Brett

The evolution of pathogens presents a paradox. Pathogenic species are often absolutely dependent on their host species for their propagation through evolutionary time, yet the pathogenic lifestyle...

SseL Is a Salmonella-Specific Translocated Effector Integrated into the SsrB-Controlled Salmonella Pathogenicity Island 2 Type III Secretion System▿

Coombes, Brian K., Lowden, Michael J., Bishop, Jennifer L., Wickham, Mark E., Brown, Nat F., Duong, Nancy, ...

Bacterial pathogens use horizontal gene transfer to acquire virulence factors that influence host colonization, alter virulence traits, and ultimately shape the outcome of disease following...

Repression of Intracellular Virulence Factors in Salmonella by the Hha and YdgT Nucleoid-Associated Proteins▿

Silphaduang, Umaporn, Mascarenhas, Mariola, Karmali, Mohamed, Coombes, Brian K.

The Hha/YmoA family of nucleoid-associated proteins is involved in gene regulation in enterobacteria. In Salmonella enterica serovar Typhimurium, virulence genes required for intracellular growth are...

Molecular Analysis as an Aid To Assess the Public Health Risk of Non-O157 Shiga Toxin-Producing Escherichia coli Strains▿ ‡

Coombes, Brian K., Wickham, Mark E., Mascarenhas, Mariola, Gruenheid, Samantha, Finlay, B. Brett, Karmali, Mohamed A.

Shiga toxin-producing Escherichia coli (STEC) strains are commensal bacteria in cattle with high potential for environmental and zoonotic transmission to humans. Although O157:H7 is the most common...

Salmonella enterica Serovar Senftenberg Human Clinical Isolates Lacking SPI-1▿

Hu, Qinghua, Coburn, Bryan, Deng, Wanyin, Li, Yuling, Shi, Xiaolu, Lan, Quanxue, ...

Nontyphoidal Salmonella species cause gastrointestinal disease worldwide. The prevailing theory of Salmonella enteropathogenesis is that bacterial invasion of the intestinal epithelium is essential...

FimH Adhesin of Type 1 Fimbriae Is a Potent Inducer of Innate Antimicrobial Responses Which Requires TLR4 and Type 1 Interferon Signalling

Ashkar, Ali A., Mossman, Karen L., Coombes, Brian K., Gyles, Carlton L., Mackenzie, Randy

Components of bacteria have been shown to induce innate antiviral immunity via Toll-like receptors (TLRs). We have recently shown that FimH, the adhesin portion of type 1 fimbria, can induce the...

Pathogenic adaptation of intracellular bacteria by rewiring a cis-regulatory input function

Osborne, Suzanne E., Walthers, Don, Tomljenovic, Ana M., Mulder, David T., Silphaduang, Uma, Duong, Nancy, ...

The acquisition of DNA by horizontal gene transfer enables bacteria to adapt to previously unexploited ecological niches. Although horizontal gene transfer and mutation of protein-coding sequences...

Role of RpoS in the Virulence of Citrobacter rodentium▿

Dong, Tao, Coombes, Brian K., Schellhorn, Herb E.

Citrobacter rodentium is a mouse enteropathogen that is closely related to Escherichia coli and causes severe colonic hyperplasia and bloody diarrhea. C. rodentium infection requires expression of...

Interleukin-15 and NK1.1+ Cells Provide Innate Protection against Acute Salmonella enterica Serovar Typhimurium Infection in the Gut and in Systemic Tissues▿

Ashkar, Ali A., Reid, Sarah, Verdu, Elena F., Zhang, Kun, Coombes, Brian K.

Control of bacterial colonization at mucosal surfaces depends on rapid activation of the innate immune system. Interleukin-15 (IL-15) directs the development, maturation, and function of a population...

Salmonella-Containing Vacuoles Display Centrifugal Movement Associated with Cell-to-Cell Transfer in Epithelial Cells▿

Szeto, Jason, Namolovan, Anton, Osborne, Suzanne E., Coombes, Brian K., Brumell, John H.

Intracellular Salmonella enterica serovar Typhimurium (serovar Typhimurium) occupies a Salmonella-containing vacuole (SCV) where bacterial effector proteins are secreted into the host cell using type...