Fangming Lin

Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia (2008)

Patel, Vishal, Li, Ling, Cobo-Stark, Patricia, Shao, Xinli, Somlo, Stefan, Lin, Fangming, ...

Polycystic kidney disease (PKD) is an inherited disorder that is characterized by the accumulation of cysts in the renal parenchyma and progressive decline in renal function. Recent studies suggest...

Kidney-specific inactivation of the KIF3A subunit of kinesin-II inhibits renal ciliogenesis and produces polycystic kidney disease

Lin, Fangming, Hiesberger, Thomas, Cordes, Kimberly, Sinclair, Angus M., Goldstein, Lawrence S. B., Somlo, Stefan, ...

Polycystic kidney disease (PKD) is the most common genetic cause of renal failure in humans. Several proteins that are encoded by genes associated with PKD have recently been identified in primary...

Loss of NFAT5 results in renal atrophy and lack of tonicity-responsive gene expression

López-Rodríguez, Cristina, Antos, Christopher L., Shelton, John M., Richardson, James A., Lin, Fangming, Novobrantseva, Tatiana I., ...

The transcription factor NFAT5/TonEBP, a member of the NFAT/Rel family of transcription factors, has been implicated in diverse cellular responses, including the response to osmotic stress,...

Intrarenal cells, not bone marrow–derived cells, are the major source for regeneration in postischemic kidney

Lin, Fangming, Moran, Ashley, Igarashi, Peter

Ischemic injury to the kidney produces acute tubular necrosis and apoptosis followed by tubular regeneration and recovery of renal function. Although mitotic cells are present in the tubules of...

Kidney-specific inactivation of the KIF3A subunit of kinesin-II inhibits renal ciliogenesis and produces polycystic kidney disease

Lin, Fangming, Hiesberger, Thomas, Cordes, Kimberly, Sinclair, Angus M., Goldstein, Lawrence S. B., Somlo, Stefan, ...

Polycystic kidney disease (PKD) is the most common genetic cause of renal failure in humans. Several proteins that are encoded by genes associated with PKD have recently been identified in primary...

Loss of NFAT5 results in renal atrophy and lack of tonicity-responsive gene expression

López-Rodríguez, Cristina, Antos, Christopher L., Shelton, John M., Richardson, James A., Lin, Fangming, Novobrantseva, Tatiana I., ...

The transcription factor NFAT5/TonEBP, a member of the NFAT/Rel family of transcription factors, has been implicated in diverse cellular responses, including the response to osmotic stress,...

Intrarenal cells, not bone marrow–derived cells, are the major source for regeneration in postischemic kidney

Lin, Fangming, Moran, Ashley, Igarashi, Peter

Ischemic injury to the kidney produces acute tubular necrosis and apoptosis followed by tubular regeneration and recovery of renal function. Although mitotic cells are present in the tubules of...