Human Proteinpedia enables sharing of human protein data (2008)
Mathivanan, S., Ahmed, M., Ahn, N. G., Alexandre, H., Amanchy, R., Andrews, P. C., ...
Inactivation of miR-34a by aberrant CpG methylation in multiple types of cancer (2008)
Lodygin, D., Tarasov, V., Epanchintsev, A., Berking, C., Knyazeva, T., Körner, H., ...
Large-scale identification of c-MYC-associated proteins using a combined TAP/MudPIT approach (2007)
Koch, H. B., Zhang, R., Verdoodt, B., Bailey, A., Zhang, C. D., Yates, J. R., ...
c-MYC delays prometaphase by direct transactivation of MAD2 and BubR1 (2007)
Menssen, A., Epanchintsev, A., Lodygin, D., Rezaei, N., Jung, P., Verdoodt, B., ...
Induction of Cullin 7 by DNA damage attenuates p53 function (2007)
Jung, P., Verdoodt, B., Bailey, A., Yates, J. R., Menssen, A., Hermeking, H.
Characterization of 14-3-3sigma dimerization determinants (2006)
Verdoodt, B., Benzinger, A., Popowicz, G. M., Holak, T. A., Hermeking, H.
Targeted proteomic analysis of 14-3-3 sigma, a p53 effector commonly silenced in cancer (2005)
Benzinger,A., Muster,N., Koch,H. B., Yates,J. R., Hermeking,H.
Functional epigenomics identifies genes frequently silenced in prostate cancer (2005)
Lodygin,D., Epanchintsev,A., Menssen,A., Diebold,J., Hermeking,H.
Mhawech,P., Benz,A., Cerato,C., Greloz,V., Assaly,M., Desmond,J. C., ...
Functional epigenomics identifies genes frequently silenced in prostate cancer (2005)
Lodygin, D., Epanchintsev, A., Menssen, A., Diebold, J., Hermeking, H.
Targeted proteomic analysis of 14-3-3 sigma, a p53 effector commonly silenced in cancer (2005)
Benzinger, A., Muster, N., Koch, H. B., Yates, J. R., Hermeking, H.
Serial analysis of gene expression and cancer (2003)
Serial analysis of gene expression has been widely used to characterize gene expression patterns associated with tumor formation. These studies resulted in the identification of tumor-specific...
Lodygin,D., Menssen,A., Hermeking,H.
Using microarray analysis, we have detected downregulation of several components of the cGMP signaling pathway during replicative senescence of primary human diploid fibroblasts (HDFs). Therefore,...
Untergasser,G., Koch,K. B., Menssen,A., Hermeking,H.
Evasion of cellular senescence is required for the immortal phenotype of tumor cells. The tumor suppressor genes p16(INK4A), pRb, and p53 have been implicated in the induction of cellular senescence....
To identify target genes of the oncogenic transcription factor c-MYC, serial analysis of gene expression (SAGE) was performed after adenoviral expression of c-MYC in primary human umbilical vein...
Untergasser, G., Koch, K. B., Menssen, A., Hermeking, H.
Evasion of cellular senescence is required for the immortal phenotype of tumor cells. The tumor suppressor genes p16(INK4A), pRb, and p53 have been implicated in the induction of cellular senescence....
To identify target genes of the oncogenic transcription factor c-MYC, serial analysis of gene expression (SAGE) was performed after adenoviral expression of c-MYC in primary human umbilical vein...
Lodygin, D., Menssen, A., Hermeking, H.
Using microarray analysis, we have detected downregulation of several components of the cGMP signaling pathway during replicative senescence of primary human diploid fibroblasts (HDFs). Therefore,...
Hermeking, H, Wolf, D A, Kohlhuber, F, Dickmanns, A, Billaud, M, Fanning, E, ...
Stably transfected NIH 3T3-L1 mouse fibroblasts (L1 cells) expressing the simian virus 40 large tumor antigen (LTAg) maintain c-myc expression and proliferation in low serum, whereas cells expressing...
Hermeking, H, Wolf, D A, Kohlhuber, F, Dickmanns, A, Billaud, M, Fanning, E, ...
Stably transfected NIH 3T3-L1 mouse fibroblasts (L1 cells) expressing the simian virus 40 large tumor antigen (LTAg) maintain c-myc expression and proliferation in low serum, whereas cells expressing...
Köster, R., Blatt, L. M., Streubert, M., Zietz, C., Hermeking, H., Brysch, W., ...
Platelet-derived growth factor-B (PDGF-B) is a potent paracrine-acting mitogen in Kaposi's sarcoma (KS) lesions. Interferon-alpha is widely used for clinical treatment of KS. Here we show that...