Hans Lassmann

Mitochondrial changes within axons in multiple sclerosis (2009)

Mahad, Don J., Ziabreva, Iryna, Campbell, Graham, Lax, Nichola, White, Katherine, Hanson, Peter S., ...

Multiple sclerosis is the most common cause of non-traumatic neurological impairment in young adults. An energy deficient state has been implicated in the degeneration of axons, the pathological...

The relation between inflammation and neurodegeneration in multiple sclerosis brains (2009)

Frischer, Josa M., Bramow, Stephan, Dal-Bianco, Assunta, Lucchinetti, Claudia F., Rauschka, Helmut, Schmidbauer, Manfred, ...

Some recent studies suggest that in progressive multiple sclerosis, neurodegeneration may occur independently from inflammation. The aim of our study was to analyse the interdependence of...

Diagnosis of inflammatory demyelination in biopsy specimens: a practical approach (2008)

Kuhlmann, Tanja, Lassmann, Hans, Brück, Wolfgang

Multiple sclerosis is the most frequent demyelinating disease in adults. It is characterized by demyelination, inflammation, gliosis and a variable loss of axons. Clinically and histologically, it...

Lesion genesis in a subset of patients with multiple sclerosis: a role for innate immunity? (2007)

Marik, Christina, Felts, Paul A., Bauer, Jan, Lassmann, Hans, Smith, Kenneth J.

Lesions obtained early in the course of multiple sclerosis (MS) have been studied immunocytochemically, and compared with the early stages of the experimental lesion induced in rats by the...

Multiple sclerosis: T-cell receptor expression in distinct brain regions (2007)

Junker, Andreas, Ivanidze, Jana, Malotka, Joachim, Eiglmeier, Ingrid, Lassmann, Hans, Wekerle, Hartmut, ...

Multiple sclerosis (MS) is an inflammatory demyelinating disease where T cells attack the brain and the spinal cord. It is known that often particular T-cell clones are expanded in the target tissue,...

Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis (2007)

Roemer, Shanu F., Parisi, Joseph E., Lennon, Vanda A., Benarroch, Eduardo E., Lassmann, Hans, Bruck, Wolfgang, ...

Neuromyelitis optica (NMO) is an inflammatory demyelinating disease that typically affects optic nerves and spinal cord. Its pathogenic relationship to multiple sclerosis (MS) is uncertain. Unlike...

Autologous haematopoietic stem cell transplantation fails to stop demyelination and neurodegeneration in multiple sclerosis (2007)

Metz, Imke, Lucchinetti, Claudia F., Openshaw, Harry, Garcia-Merino, Antonio, Lassmann, Hans, Freedman, Marc S., ...

The present study analyses autopsy material from five multiple sclerosis patients who received autologous stem cell transplantation. A total of 53 white matter lesions were investigated using routine...

Autologous haematopoietic stem cell transplantation fails to stop demyelination and neurodegeneration in multiple sclerosis (2007)

Metz, Imke, Lucchinetti, Claudia F., Openshaw, Harry, Garcia-Merino, Antonio, Lassmann, Hans, Freedman, Marc S., ...

The present study analyses autopsy material from five multiple sclerosis patients who received autologous stem cell transplantation. A total of 53 white matter lesions were investigated using routine...

Pattern-specific loss of aquaporin-4 immunoreactivity distinguishes neuromyelitis optica from multiple sclerosis (2007)

Roemer, Shanu F., Parisi, Joseph E., Lennon, Vanda A., Benarroch, Eduardo E., Lassmann, Hans, Bruck, Wolfgang, ...

Neuromyelitis optica (NMO) is an inflammatory demyelinating disease that typically affects optic nerves and spinal cord. Its pathogenic relationship to multiple sclerosis (MS) is uncertain. Unlike...

Remyelination is extensive in a subset of multiple sclerosis patients (2006)

Patrikios, Peter, Stadelmann, Christine, Kutzelnigg, Alexandra, Rauschka, Helmut, Schmidbauer, Manfred, Laursen, Henning, ...

Although spontaneous remyelination does occur in multiple sclerosis lesions, its extent within the global population with this disease is presently unknown. We have systematically analysed the...

Understanding pathogenesis and therapy of multiple sclerosis via animal models: 70 years of merits and culprits in experimental autoimmune encephalomyelitis research (2006)

Gold, Ralf, Linington, Christopher, Lassmann, Hans

In view of disease heterogeneity of multiple sclerosis and limited access to ex vivo specimens, different approaches must be undertaken to better understand disease pathogenesis and new therapeutic...

Remyelination is extensive in a subset of multiple sclerosis patients (2006)

Patrikios, Peter, Stadelmann, Christine, Kutzelnigg, Alexandra, Rauschka, Helmut, Schmidbauer, Manfred, Laursen, Henning, ...

Although spontaneous remyelination does occur in multiple sclerosis lesions, its extent within the global population with this disease is presently unknown. We have systematically analysed the...

Understanding pathogenesis and therapy of multiple sclerosis via animal models: 70 years of merits and culprits in experimental autoimmune encephalomyelitis research (2006)

Gold, Ralf, Linington, Christopher, Lassmann, Hans

In view of disease heterogeneity of multiple sclerosis and limited access to ex vivo specimens, different approaches must be undertaken to better understand disease pathogenesis and new therapeutic...

Cortical demyelination and diffuse white matter injury in multiple sclerosis (2005)

Kutzelnigg, Alexandra, Lucchinetti, Claudia F., Stadelmann, Christine, Brück, Wolfgang, Rauschka, Helmut, Bergmann, Markus, ...

Focal demyelinated plaques in white matter, which are the hallmark of multiple sclerosis pathology, only partially explain the patient's clinical deficits. We thus analysed global brain pathology in...

Tissue preconditioning may explain concentric lesions in Balo's type of multiple sclerosis (2005)

Stadelmann, Christine, Ludwin, Sam, Tabira, Takeshi, Guseo, Andras, Lucchinetti, Claudia F., Leel-Össy, Lorant, ...

Lesions of Baló's concentric sclerosis are characterized by alternating layers of myelinated and demyelinated tissue. The reason for concentric demyelination in this variant of multiple sclerosis is...

Neuropathology of CNS disease in Langerhans cell histiocytosis (2005)

Grois, Nicole, Prayer, Daniela, Prosch, Helmut, Lassmann, Hans

CNS involvement in Langerhans cell histiocytosis (LCH) is a rare but potentially devastating disorder. Different types of involvement have been described by MRI. CNS changes can have space-occupying...

Neuropathology of white matter disease in Leber's hereditary optic neuropathy (2005)

Kovács, Gábor G., Höftberger, Romana, Majtényi, Katalin, Horváth, Rita, Barsi, Péter, Komoly, Sámuel, ...

Leber's hereditary optic neuropathy (LHON) is associated with point mutations in the mitochondrial DNA (mtDNA), coding for a mitochondrial respiratory chain complex I subunit. It is characterized by...

Neuropathology of CNS disease in Langerhans cell histiocytosis (2005)

Grois, Nicole, Prayer, Daniela, Prosch, Helmut, Lassmann, Hans

Summary CNS involvement in Langerhans cell histiocytosis (LCH) is a rare but potentially devastating disorder. Different types of involvement have been described by MRI. CNS changes can have...

Tissue preconditioning may explain concentric lesions in Balo's type of multiple sclerosis (2005)

Stadelmann, Christine, Ludwin, Sam, Tabira, Takeshi, Guseo, Andras, Lucchinetti, Claudia F., Leel-Össy, Lorant, ...

Summary Lesions of Baló's concentric sclerosis are characterized by alternating layers of myelinated and demyelinated tissue. The reason for concentric demyelination in this variant of multiple...

Cortical demyelination and diffuse white matter injury in multiple sclerosis (2005)

Kutzelnigg, Alexandra, Lucchinetti, Claudia F., Stadelmann, Christine, Brück, Wolfgang, Rauschka, Helmut, Bergmann, Markus, ...

Focal demyelinated plaques in white matter, which are the hallmark of multiple sclerosis pathology, only partially explain the patient's clinical deficits. We thus analysed global brain pathology in...

Modelling paraneoplastic CNS disease: T-cells specific for the onconeuronal antigen PNMA1 mediate autoimmune encephalomyelitis in the rat (2004)

Pellkofer, Hannah, Schubart, Anna S., Höftberger, Romana, Schutze, Nadine, Pagany, Maria, Schüller, Martina, ...

Summary Antibodies directed against onconeuronal antigens provide a specific diagnostic marker for paraneoplastic neurological syndromes (PNS) and suggest that these autoantigens are targeted during...

Neuropathology of white matter disease in Leber's hereditary optic neuropathy (2004)

Kovács, Gábor G., Höftberger, Romana, Majtényi, Katalin, Horváth, Rita, Barsi, Péter, Komoly, Sámuel, ...

Summary Leber's hereditary optic neuropathy (LHON) is associated with point mutations in the mitochondrial DNA (mtDNA), coding for a mitochondrial respiratory chain complex I subunit. It is...

Modelling paraneoplastic CNS disease: T-cells specific for the onconeuronal antigen PNMA1 mediate autoimmune encephalomyelitis in the rat (2004)

Pellkofer, Hannah, Schubart, Anna S., Höftberger, Romana, Schutze, Nadine, Pagany, Maria, Schüller, Martina, ...

Antibodies directed against onconeuronal antigens provide a specific diagnostic marker for paraneoplastic neurological syndromes (PNS) and suggest that these autoantigens are targeted during disease...

Modelling paraneoplastic CNS disease: T-cells specific for the onconeuronal antigen PNMA1 mediate autoimmune encephalomyelitis in the rat (2004)

Pellkofer, Hannah, Schubart, Anna S., Höftberger, Romana, Schutze, Nadine, Pagany, Maria, Schüller, Martina, ...

Summary Antibodies directed against onconeuronal antigens provide a specific diagnostic marker for paraneoplastic neurological syndromes (PNS) and suggest that these autoantigens are targeted during...

Neuropathology of white matter disease in Leber's hereditary optic neuropathy (2004)

Kovács, Gábor G., Höftberger, Romana, Majtényi, Katalin, Horváth, Rita, Barsi, Péter, Komoly, Sámuel, ...

Summary Leber's hereditary optic neuropathy (LHON) is associated with point mutations in the mitochondrial DNA (mtDNA), coding for a mitochondrial respiratory chain complex I subunit. It is...

A new paraclinical CSF marker for hypoxia-like tissue damage in multiple sclerosis lesions (2003)

Lassmann, Hans, Reindl, Markus, Rauschka, Helmut, Berger, Johannes, Aboul-Enein, Fahmy, Berger, Thomas, ...

Recent studies on the immunopathology of multiple sclerosis revealed a heterogeneity in the patterns of demyelination, suggesting interindividual differences in the mechanism responsible for myelin...

Distinct time pattern of complement activation and cytotoxic T cell response in Guillain-Barre syndrome (2003)

Wanschitz, Julia, Maier, Hans, Lassmann, Hans, Budka, Herbert, Berger, Thomas

Humoural and cellular immune mechanisms are involved in the pathogenesis of Guillain-Barré syndrome (GBS). While activation of complement has been implicated in the initiation of myelin damage, we...

Distinct time pattern of complement activation and cytotoxic T cell response in Guillain-Barre syndrome (2003)

Wanschitz, Julia, Maier, Hans, Lassmann, Hans, Budka, Herbert, Berger, Thomas

Humoural and cellular immune mechanisms are involved in the pathogenesis of Guillain–Barré syndrome (GBS). While activation of complement has been implicated in the initiation of myelin damage, we...

A new paraclinical CSF marker for hypoxia-like tissue damage in multiple sclerosis lesions (2003)

Lassmann, Hans, Reindl, Markus, Rauschka, Helmut, Berger, Johannes, Aboul-Enein, Fahmy, Berger, Thomas, ...

Recent studies on the immunopathology of multiple sclerosis revealed a heterogeneity in the patterns of demyelination, suggesting interindividual differences in the mechanism responsible for myelin...

Distinct time pattern of complement activation and cytotoxic T cell response in Guillain-Barre syndrome (2003)

Wanschitz, Julia, Maier, Hans, Lassmann, Hans, Budka, Herbert, Berger, Thomas

Humoural and cellular immune mechanisms are involved in the pathogenesis of Guillain-Barré syndrome (GBS). While activation of complement has been implicated in the initiation of myelin damage, we...

Fc receptors are critical for autoimmune inflammatory damage to the central nervous system in experimental autoimmune encephalomyelitis (2002)

Abdul-Majid, K. B., Stefferl, Andreas, Bourquin, Carole, Lassmann, Hans, Linington, Christopher, Olsson, T., ...

Multiple sclerosis (MS) is simulated by various forms of experimental autoimmune encephalomyelitis, in which T cells, antibodies, cytokines and complementary factors interact with the central nervous...

Endoplasmic reticulum stress in PLP-overexpressing transgenic rats: Gray matter oligodendrocytes are more vulnerable than white matter oligodendrocytes (2002)

Bauer, Jan, Bradl, Monika, Klein, Matthias, Leisser, M., Deckwerth, T. L., Wekerle, Hartmut, ...

Studies dealing with transport of proteins from the oligodendrocyte cell body it) the myelin sheath reveal the presence of different transport pathways. Proteolipid protein (PLP) is synthesized at...

Skeletal, cardiac and tongue muscle pathology, defective retinal transmission, and neuronal migration defects in the Largemyd mouse defines a natural model for glycosylation-deficient muscle - eye - brain disorders (2002)

Holzfeind, Paul J., Grewal, Prabhjit K., Reitsamer, Herbert A., Kechvar, Jasmin, Lassmann, Hans, Hoeger, Harald, ...

We have recently shown that a deletion in the Large gene, encoding a putative glycosyltransferase, is the molecular defect underlying the myodystrophy (previously myd; now Largemyd) mouse. Here we...

BDNF and gp145trkB in multiple sclerosis brain lesions: neuroprotective interactions between immune and neuronal cells? (2002)

Stadelmann, Christine, Kerschensteiner, Martin, Misgeld, Thomas, Brück, Wolfgang, Hohlfeld, Reinhard, Lassmann, Hans

Recent immunohistological and imaging studies emphasize the crucial role of axonal injury in determining the extent of permanent neurological deficits in patients with multiple sclerosis. We have...

A role for humoral mechanisms in the pathogenesis of Devic's neuromyelitis optica (2002)

Lucchinetti, Claudia F., Mandler, Raul N., McGavern, Dorian, Bruck, Wolfgang, Gleich, Gerald, Ransohoff, Richard M., ...

Devic’s disease [neuromyelitis optica (NMO)] is an idiopathic inflammatory demyelinating disease of the CNS, characterized by attacks of optic neuritis and myelitis. The mechanisms that...

Distribution of a calcium channel subunit in dystrophic axons in multiple sclerosis and experimental autoimmune encephalomyelitis (2001)

Kornek, Barbara, Storch, Maria K., Bauer, Jan, Djamshidian, Atbin, Weissert, Robert, Wallstroem, Erik, ...

Multiple sclerosis and experimental autoimmune encephalomyelitis (EAE) are immune-mediated diseases of the CNS. They are characterized by widespread inflammation, demyelination and a variable degree...

TNFR1 signalling is critical for the development of demyelination and the limitation of T-cell responses during immune-mediated CNS disease (2000)

Probert, Lesley, Eugster, Hans-Pietro, Akassoglou, Katerina, Bauer, Jan, Frei, Karl, Lassmann, Hans, ...

In this review we summarize the essential findings about the function of tumour necrosis factor (TNF) and its cognate receptors TNFR1 and TNFR2, and lymphotoxin α (LT-α) ligands in immune-mediated...

A quantitative analysis of oligodendrocytes in multiple sclerosis lesions: A study of 113 cases (1999)

Lucchinetti, Claudia, Brück, Wolfgang, Parisi, Joseph, Scheithauer, Bernd, Rodriguez, Moses, Lassmann, Hans

We studied quantitatively the fate of oligodendrocytes (OGs) during lesion formation in 395 lesion areas from biopsy and autopsy tissue of 113 multiple sclerosis cases. The density of OGs in multiple...

In vivo MRI and its histological correlates in acute adoptive transfer experimental allergic encephalomyelitis: Quantification of inflammation and oedema (1996)

Morrissey, Sean P., Stodal, Horst, Zettl, Uwe, Simonis, Claudia, Jung, Stefan, Kiefer, Reinhard, ...

In vivo proton MRI was carried out on a 7 Tesla system at 2–3 day intervals over 10 days in rats with adoptive transfer experimental allergic encephalomyelitis (AT-EAE), an animal model of some...

Activated Human T Cells, B Cells, and Monocytes Produce Brain-derived Neurotrophic Factor In Vitro and in Inflammatory Brain Lesions: A Neuroprotective Role of Inflammation?

Kerschensteiner, Martin, Gallmeier, Eike, Behrens, Lüder, Leal, Vivian Vargas, Misgeld, Thomas, Klinkert, Wolfgang E.F., ...

Brain-derived neurotrophic factor (BDNF) has potent effects on neuronal survival and plasticity during development and after injury. In the nervous system, neurons are considered the major cellular...

Clonal Expansions of Cd8+ T Cells Dominate the T Cell Infiltrate in Active Multiple Sclerosis Lesions as Shown by Micromanipulation and Single Cell Polymerase Chain Reaction

Babbe, Holger, Roers, Axel, Waisman, Ari, Lassmann, Hans, Goebels, Norbert, Hohlfeld, Reinhard, ...

Clonal composition and T cell receptor (TCR) repertoire of CD4+ and CD8+ T cells infiltrating actively demyelinating multiple sclerosis (MS) lesions were determined with unprecedented resolution at...

Vaccination with DNA Encoding an Immunodominant Myelin Basic Protein Peptide Targeted to Fc of Immunoglobulin G Suppresses Experimental Autoimmune Encephalomyelitis

Lobell, Anna, Weissert, Robert, Storch, Maria K., Svanholm, Cecilia, De Graaf, Katrien L., Lassmann, Hans, ...

We explore here if vaccination with DNA encoding an autoantigenic peptide can suppress autoimmune disease. For this purpose we used experimental autoimmune encephalomyelitis (EAE), which is an...

Map1b Is Required for Axon Guidance and Is Involved in the Development of the Central and Peripheral Nervous System

Meixner, Arabella, Haverkamp, Silke, Wässle, Heinz, Führer, Susanne, Thalhammer, Johann, Kropf, Nina, ...

Microtubule-associated proteins such as MAP1B have long been suspected to play an important role in neuronal differentiation, but proof has been lacking. Previous MAP1B gene targeting studies yielded...

Mutation in the Scyl1 gene encoding amino-terminal kinase-like protein causes a recessive form of spinocerebellar neurodegeneration

Schmidt, Wolfgang M, Kraus, Cornelia, Höger, Harald, Hochmeister, Sonja, Oberndorfer, Felicitas, Branka, Manuela, ...

Here, we show that the murine neurodegenerative disease mdf (autosomal recessive mouse mutant ‘muscle deficient') is caused by a loss-of-function mutation in Scyl1, disrupting the expression of...

Mitochondrial defects in acute multiple sclerosis lesions

Mahad, Don, Ziabreva, Iryna, Lassmann, Hans, Turnbull, Douglas

Multiple sclerosis is a chronic inflammatory disease, which leads to focal plaques of demyelination and tissue injury in the CNS. The structural and immunopathological patterns of demyelination...

B Lymphocytes Producing Demyelinating Autoantibodies: Development and Function in Gene-targeted Transgenic Mice

Litzenburger, Tobias, Fässler, Reinhard, Bauer, Jan, Lassmann, Hans, Linington, Christopher, Wekerle, Hartmut, ...

We studied the cellular basis of self tolerance of B cells specific for brain autoantigens using transgenic mice engineered to produce high titers of autoantibodies against the myelin oligodendrocyte...

Diagnosis of inflammatory demyelination in biopsy specimens: a practical approach

Kuhlmann, Tanja, Lassmann, Hans, Brück, Wolfgang

Multiple sclerosis is the most frequent demyelinating disease in adults. It is characterized by demyelination, inflammation, gliosis and a variable loss of axons. Clinically and histologically, it...

After Injection into the Striatum, in Vitro-Differentiated Microglia- and Bone Marrow-Derived Dendritic Cells Can Leave the Central Nervous System via the Blood Stream

Hochmeister, Sonja, Zeitelhofer, Manuel, Bauer, Jan, Nicolussi, Eva-Maria, Fischer, Marie-Therese, Heinke, Bernhard, ...

The prototypic migratory trail of tissue-resident dendritic cells (DCs) is via lymphatic drainage. Since the central nervous system (CNS) lacks classical lymphatic vessels, and antigens and cells...

The relation between inflammation and neurodegeneration in multiple sclerosis brains

Frischer, Josa M., Bramow, Stephan, Dal-Bianco, Assunta, Lucchinetti, Claudia F., Rauschka, Helmut, Schmidbauer, Manfred, ...

Some recent studies suggest that in progressive multiple sclerosis, neurodegeneration may occur independently from inflammation. The aim of our study was to analyse the interdependence of...

Contactin-2/TAG-1-directed autoimmunity is identified in multiple sclerosis patients and mediates gray matter pathology in animals

Derfuss, Tobias, Parikh, Khyati, Velhin, Sviataslau, Braun, Magdalena, Mathey, Emily, Krumbholz, Markus, ...

Gray matter pathology is increasingly recognized as an important feature of multiple sclerosis (MS), but the nature of the immune response that targets the gray matter is poorly understood. Starting...