Hideki Katoh

Extracellular acidosis suppresses endothelial function by inhibiting store-operated Ca2+ entry via non-selective cation channels (2009)

Asai, Masayoshi, Takeuchi, Kazuhiko, Saotome, Masao, Urushida, Tsuyoshi, Katoh, Hideki, Satoh, Hiroshi, ...

Aims Hypoxia, ischaemia, and exogenous chemicals can induce extracellular and intracellular acidosis, but it is not clear which of these types of acidosis affects endothelial cell function. The...

Endoplasmic reticulum Ca2+ depletion induces endothelial cell apoptosis independently of caspase-12 (2006)

Nakano, Tomoyasu, Watanabe, Hiroshi, Ozeki, Mariko, Asai, Masayoshi, Katoh, Hideki, Satoh, Hiroshi, ...

Objective: Apoptosis of endothelial cells is considered an initial step in the development of atherosclerosis. Recent studies have indicated that depletion of the endoplasmic reticulum (ER) Ca2+...

Heterogeneity and underlying mechanism for inotropic action of endothelin-1 in rat ventricular myocytes

Katoh, Hideki, Terada, Hajime, Iimuro, Masaru, Sugiyama, Shiho, Qing, Ke, Satoh, Hiroshi, ...

To clarify the mechanisms underlying the positive inotropic action of endothelin-1 (ET-1), we investigated the effect of ET-1 on twitch cell shortening and the Ca2+ transient in rat isolated...