Granhall, Charlotte, Park, Hee-Bok, Fakhrai-Rad, Hossein, Luthman, Holger
Abstract is not available
Identification of rat susceptibility loci for adjuvant-oil-induced arthritis
Lorentzen, Johnny C., Glaser, Anna, Jacobsson, Lena, Galli, Joakim, Fakhrai-rad, Hossein, Klareskog, Lars, ...
One intradermal injection of incomplete Freund’s adjuvant-oil induces a T cell-mediated inflammatory joint disease in DA rats. Susceptibility genes for oil-induced arthritis (OIA) are located both...
SNP Discovery in Pooled Samples With Mismatch Repair Detection
Fakhrai-Rad, Hossein, Zheng, Jianbiao, Willis, Thomas D., Wong, Kee, Suyenaga, Kent, Moorhead, Martin, ...
A targeted discovery effort is required to identify low frequency single nucleotide polymorphisms (SNPs) in human coding and regulatory regions. We here describe combining mismatch repair detection...
Faham, Malek, Zheng, Jianbiao, Moorhead, Martin, Fakhrai-Rad, Hossein, Namsaraev, Eugeni, Wong, Kee, ...
Identification of the genetic basis of common disease may require comprehensive sequence analysis of coding regions and regulatory elements in patients and controls to find genetic effects caused by...
Identification of rat susceptibility loci for adjuvant-oil-induced arthritis
Lorentzen, Johnny C., Glaser, Anna, Jacobsson, Lena, Galli, Joakim, Fakhrai-rad, Hossein, Klareskog, Lars, ...
One intradermal injection of incomplete Freund’s adjuvant-oil induces a T cell-mediated inflammatory joint disease in DA rats. Susceptibility genes for oil-induced arthritis (OIA) are located both...
SNP Discovery in Pooled Samples With Mismatch Repair Detection
Fakhrai-Rad, Hossein, Zheng, Jianbiao, Willis, Thomas D., Wong, Kee, Suyenaga, Kent, Moorhead, Martin, ...
A targeted discovery effort is required to identify low frequency single nucleotide polymorphisms (SNPs) in human coding and regulatory regions. We here describe combining mismatch repair detection...
Faham, Malek, Zheng, Jianbiao, Moorhead, Martin, Fakhrai-Rad, Hossein, Namsaraev, Eugeni, Wong, Kee, ...
Identification of the genetic basis of common disease may require comprehensive sequence analysis of coding regions and regulatory elements in patients and controls to find genetic effects caused by...
Granhall, Charlotte, Park, Hee-Bok, Fakhrai-Rad, Hossein, Luthman, Holger
Niddm1i, a 16-Mb locus within the major diabetes QTL in the diabetic GK rat, causes impaired glucose tolerance in the congenic NIDDM1I strain. Niddm1i is homologous to both human and mouse regions...