I. Nonaka

Publication List Details

Period

1981 - 2008

Number

30

Co-Authors

Making the Most of Your Company's Knowledge: A Strategic Framework (2001)

Georg Vonkrogh, Manfred Aben, I. Nonaka

This paper develops a framework of four strategies for managing knowledge. Companies can leverage their knowledge throughout the organisation, expand their knowledge further based on existing...

A firm as a knowledge-creating entity: a new perspective on the theory of the firm (2000)

Nonaka, I, Toyama, R, Nagata, A

The knowledge-based view of the firm views a firm as a knowledge-creating entity, and argues that knowledge and the capability to create and utilize such knowledge are the most important source of a...

Possible influences on the expression of X chromosome-linked dystrophin abnormalities by heterozygosity for autosomal recessive Fukuyama congenital muscular dystrophy.

Beggs, A H, Neumann, P E, Arahata, K, Arikawa, E, Nonaka, I, Anderson, M S, ...

Abnormalities of dystrophin, a cytoskeletal protein of muscle and nerve, are generally considered specific for Duchenne and Becker muscular dystrophy. However, several patients have recently been...

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcripts.

Chomyn, A, Martinuzzi, A, Yoneda, M, Daga, A, Hurko, O, Johns, D, ...

The pathogenetic mechanism of the mitochondrial tRNA(LeuUUR) gene mutation responsible for the MELAS (mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes) syndrome was...

Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction.

Hayashi, J, Ohta, S, Kikuchi, A, Takemitsu, M, Goto, Y, Nonaka, I

Mutant mitochondrial DNA with large-scale deletions (delta-mtDNA) has been frequently observed in patients with chronic progressive external ophthalmoplegia (CPEO), a subgroup of the mitochondrial...

Dystrophin diagnosis: comparison of dystrophin abnormalities by immunofluorescence and immunoblot analyses.

Arahata, K, Hoffman, E P, Kunkel, L M, Ishiura, S, Tsukahara, T, Ishihara, T, ...

Immunoblot characterization and immunofluorescence localization of dystrophin are presented for 76 human patients with various neuromuscular diseases. Normal dystrophin (shown by immunoblotting) was...

Mild deficiency of dystrophin-associated proteins in Becker muscular dystrophy patients having in-frame deletions in the rod domain of dystrophin.

Matsumura, K, Nonaka, I, Tomé, F M, Arahata, K, Collin, H, Leturcq, F, ...

The dystrophin-glycoprotein complex spans the sarcolemma to provide a linkage between the subsarcolemmal cytoskeleton and the extracellular matrix in skeletal muscle. In Duchenne muscular dystrophy...

Possible influences on the expression of X chromosome-linked dystrophin abnormalities by heterozygosity for autosomal recessive Fukuyama congenital muscular dystrophy.

Beggs, A H, Neumann, P E, Arahata, K, Arikawa, E, Nonaka, I, Anderson, M S, ...

Abnormalities of dystrophin, a cytoskeletal protein of muscle and nerve, are generally considered specific for Duchenne and Becker muscular dystrophy. However, several patients have recently been...

MELAS mutation in mtDNA binding site for transcription termination factor causes defects in protein synthesis and in respiration but no change in levels of upstream and downstream mature transcripts.

Chomyn, A, Martinuzzi, A, Yoneda, M, Daga, A, Hurko, O, Johns, D, ...

The pathogenetic mechanism of the mitochondrial tRNA(LeuUUR) gene mutation responsible for the MELAS (mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes) syndrome was...

Introduction of disease-related mitochondrial DNA deletions into HeLa cells lacking mitochondrial DNA results in mitochondrial dysfunction.

Hayashi, J, Ohta, S, Kikuchi, A, Takemitsu, M, Goto, Y, Nonaka, I

Mutant mitochondrial DNA with large-scale deletions (delta-mtDNA) has been frequently observed in patients with chronic progressive external ophthalmoplegia (CPEO), a subgroup of the mitochondrial...

Dystrophin diagnosis: comparison of dystrophin abnormalities by immunofluorescence and immunoblot analyses.

Arahata, K, Hoffman, E P, Kunkel, L M, Ishiura, S, Tsukahara, T, Ishihara, T, ...

Immunoblot characterization and immunofluorescence localization of dystrophin are presented for 76 human patients with various neuromuscular diseases. Normal dystrophin (shown by immunoblotting) was...

Mild deficiency of dystrophin-associated proteins in Becker muscular dystrophy patients having in-frame deletions in the rod domain of dystrophin.

Matsumura, K, Nonaka, I, Tomé, F M, Arahata, K, Collin, H, Leturcq, F, ...

The dystrophin-glycoprotein complex spans the sarcolemma to provide a linkage between the subsarcolemmal cytoskeleton and the extracellular matrix in skeletal muscle. In Duchenne muscular dystrophy...

Analysis of lymphoproliferative cytokines produced by thymic myoid cells.

Iwakami, N, Kikuchi, A, Kunishita, T, Yamamoto, H, Nonaka, I, Kamo, I

Lymphoproliferative activities produced by cloned thymic myoid cell 871207B were analysed by immunological and biochemical methods. The lymphoproliferative activities were separated into two...

Immunological and biochemical characterization of biglycan-like haemopoietic factor.

Kikuchi, A, Iwakami, N, Takahashi, K, Suzuki, J, Kondo, J, Yamada, E, ...

Immunological and biochemical characteristics of a 100,000 MW biglycan-like haemopoietic factor, purified from thymic myoid cells 871207B, were studied to distinguish them from macrophage...

Characterization of a macrophage lineage cell colony-stimulating factor produced by thymic myoid cells.

Kamo, I, Kunishita, T, Kikuchi, A, Nonaka, I, Komiyama, A

Thymic myoid cells produced macrophage lineage cell stimulatory factors. Activities were separated into two factors on DEAE-Sepharose CL-6B chromatography: one eluted at lower concentrations of NaCl...

Sarcoglycan complex is selectively lost in dystrophic hamster muscle.

Mizuno, Y., Noguchi, S., Yamamoto, H., Yoshida, M., Nonaka, I., Hirai, S., ...

We recently reported that the dystrophin-associated glycoprotein (DAG) complex is biochemically divided into two subcomplexes: one is the dystroglycan complex comprised of 156DAG and 43DAG and the...

Abnormal increases of lysosomal cysteinine proteinases in rimmed vacuoles in the skeletal muscle.

Ii, K., Hizawa, K., Nonaka, I., Sugita, H., Kominami, E., Katunuma, N.

Skeletal muscle obtained from a patient with distal myopathy with rimmed vacuole formation (DMRV) was examined by a new direct labeled antibody enzyme method of immunohistochemistry. Abnormal...

Three new mutations in patients with myophosphorylase deficiency (McArdle disease).

Tsujino, S., Shanske, S., Nonaka, I., Eto, Y., Mendell, J. R., Fenichel, G. M., ...

We report three new mutations in patients with myophosphorylase deficiency (McArdle disease). A splice-junction mutation (G-to-A transition at the 5' end of intron 14) and a missense mutation (CTG to...