J. P. Kehrer

Publication List Details

Number

10

Co-Authors

Oxidation state of tissue thiol groups and content of protein carbonyl groups in chickens with inherited muscular dystrophy.

Murphy, M E, Kehrer, J P

Indirect evidence suggests that oxidative stress may play a role in the pathogenesis of inherited muscular dystrophy, but the significance and precise extent of this contribution is poorly...

Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal.

Bojes, H K, Datta, K, Xu, J, Chin, A, Simonian, P, Nuñez, G, ...

Bcl-xL and bax are bcl-2-related genes whose protein products either inhibit or promote apoptosis. Oxidative damage, including the loss of glutathione, has been implicated in the induction of...

The 5-lipoxygenase-activating protein (FLAP) inhibitor, MK886, induces apoptosis independently of FLAP.

Datta, K, Biswal, S S, Kehrer, J P

The ability of various inhibitors of lipoxygenase (LOX) enzymes and 5-lipoxygenase-activating protein (FLAP) to induce apoptosis has implicated these pathways in the mechanism(s) of this form of cell...

Heat-shock protein 70 antisense oligomers enhance proteasome inhibitor-induced apoptosis.

Robertson, J D, Datta, K, Biswal, S S, Kehrer, J P

Recent evidence supports a role for heat-shock protein 70 (hsp70) and the 26 S proteasome in regulating apoptosis, although the precise nature of their involvement is not known. In the present study,...

Inhibition of peroxisome-proliferator-activated receptor (PPAR)alpha by MK886.

Kehrer, J P, Biswal, S S, La, E, Thuillier, P, Datta, K, Fischer, S M, ...

Although MK886 was originally identified as an inhibitor of 5-lipoxygenase activating protein (FLAP), recent data demonstrate that this activity does not underlie its ability to induce apoptosis...

Oxidation state of tissue thiol groups and content of protein carbonyl groups in chickens with inherited muscular dystrophy.

Murphy, M E, Kehrer, J P

Indirect evidence suggests that oxidative stress may play a role in the pathogenesis of inherited muscular dystrophy, but the significance and precise extent of this contribution is poorly...

Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal.

Bojes, H K, Datta, K, Xu, J, Chin, A, Simonian, P, Nuñez, G, ...

Bcl-xL and bax are bcl-2-related genes whose protein products either inhibit or promote apoptosis. Oxidative damage, including the loss of glutathione, has been implicated in the induction of...

The 5-lipoxygenase-activating protein (FLAP) inhibitor, MK886, induces apoptosis independently of FLAP.

Datta, K, Biswal, S S, Kehrer, J P

The ability of various inhibitors of lipoxygenase (LOX) enzymes and 5-lipoxygenase-activating protein (FLAP) to induce apoptosis has implicated these pathways in the mechanism(s) of this form of cell...

Heat-shock protein 70 antisense oligomers enhance proteasome inhibitor-induced apoptosis.

Robertson, J D, Datta, K, Biswal, S S, Kehrer, J P

Recent evidence supports a role for heat-shock protein 70 (hsp70) and the 26 S proteasome in regulating apoptosis, although the precise nature of their involvement is not known. In the present study,...

Inhibition of peroxisome-proliferator-activated receptor (PPAR)alpha by MK886.

Kehrer, J P, Biswal, S S, La, E, Thuillier, P, Datta, K, Fischer, S M, ...

Although MK886 was originally identified as an inhibitor of 5-lipoxygenase activating protein (FLAP), recent data demonstrate that this activity does not underlie its ability to induce apoptosis...