Jill Dixon

Predictors of Mortality and Morbidity in Clostridium Difficile Infection (2009)

Jill Dixon, Brian F. Menezes

Background: Clostridium Difficile (CD) is implicated in 20 to 30% of patients with antibiotic-associated diarrhoea, in 50 to 70% of those with antibiotic-associated colitis and in more than 90% of...

Integration of IRF6 and Jagged2 signalling is essential for controlling palatal adhesion and fusion competence (2009)

Richardson, Rebecca J., Dixon, Jill, Jiang, Rulang, Dixon, Michael J.

In mammals, adhesion and fusion of the palatal shelves are essential mechanisms during the development of the secondary palate; failure of these processes leads to the congenital anomaly, cleft...

Service level agreements: a framework for the quality management and improvement of central support services. Abstract: (2008)

Melany Blackwell, Jill Dixon

Service level agreements (SLAs) are becoming an increasingly popular way to manage the quality of services within organisations. Monash University is developing SLA's between central support...

The Treacher Collins syndrome (TCOF1) gene product is involved in pre-rRNA methylation (2005)

Gonzales, Bianca, Henning, Dale, So, Rolando B., Dixon, Jill, Dixon, Michael J., Valdez, Benigno C.

Treacher Collins syndrome (TCS) is characterized by defects in craniofacial development, which results from mutations in the TCOF1 gene. TCOF1 encodes the nucleolar phosphoprotein treacle, which...

Identification of mutations in TCOF1: use of molecular analysis in the pre- and postnatal diagnosis of Treacher Collins syndrome (2004)

Dixon, Jill, Ellis, Ian, Bottani, Armand, Temple, Karen, Dixon, Michael James

Treacher Collins syndrome (TCS) is an autosomal dominant disorder of facial development, which results from mutations in TCOF1. TCS comprises conductive hearing loss, hypoplasia of the mandible and...

Mutation of the gene encoding fibrillin-2 results in syndactyly in mice (2001)

Chaudhry, Shazia S., Gazzard, James, Baldock, Clair, Dixon, Jill, Rock, Matthew J., Skinner, Gail C., ...

Fibrillins are large, cysteine-rich glycoproteins that form microfibrils and play a central role in elastic fibrillogenesis. Fibrillin-1 and fibrillin-2, encoded by FBN1 on chromosome 15q21.1 and...

Increased levels of apoptosis in the prefusion neural folds underlie the craniofacial disorder, Treacher Collins syndrome (2000)

Dixon, Jill, Brakebusch, Cord, Fässler, Reinhard, Dixon, Michael J.

Treacher Collins syndrome (TCS) is an autosomal dominant disorder of human craniofacial development that results from loss-of-function mutations in the gene TCOF1. Although this gene has been...

Genetic and physical mapping of the Treacher Collins syndrome locus: refinement of the localization to chromosome 5q32-33.2 (1992)

Dixon, Michael J., Dixon, Jill, Raskova, Dagmar, Le Beau, Michelle M., Williamson, Robert, Kinger, Katherine, ...

Treacher Collins syndrome (TCOF1) is an autosomal dominant disorder of craniofacial development, the locus for which has been chromosomally localized to 5q31-34. We have isolated four hypervarlable...

Characterization of the Nucleolar Gene Product, Treacle, in Treacher Collins Syndrome

Isaac, Cynthia, Marsh, Karen L., Paznekas, William A., Dixon, Jill, Dixon, Michael J., Jabs, Ethylin Wang, ...

Treacher Collins syndrome (TCS) is an autosomal dominant disorder of craniofacial development caused by mutations in the gene TCOF1. Its gene product, treacle, consists mainly of a central repeat...

The Treacher Collins syndrome (TCOF1) gene product is involved in ribosomal DNA gene transcription by interacting with upstream binding factor

Valdez, Benigno C., Henning, Dale, So, Rolando B., Dixon, Jill, Dixon, Michael J.

Treacher Collins syndrome (TCS) is an autosomal dominant disorder characterized by an abnormality of craniofacial development that arises during early embryogenesis. TCS is caused by mutations in the...

Targeted Deletion of mek5 Causes Early Embryonic Death and Defects in the Extracellular Signal-Regulated Kinase 5/Myocyte Enhancer Factor 2 Cell Survival Pathway

Wang, Xin, Merritt, Anita J., Seyfried, Jan, Guo, Chun, Papadakis, Emmanouil S., Finegan, Katherine G., ...

To elucidate the physiological significance of MEK5 in vivo, we have examined the effect of mek5 gene elimination in mice. Heterozygous mice appear to be healthy and were fertile. However,...

Characterization of the Nucleolar Gene Product, Treacle, in Treacher Collins Syndrome

Isaac, Cynthia, Marsh, Karen L., Paznekas, William A., Dixon, Jill, Dixon, Michael J., Jabs, Ethylin Wang, ...

Treacher Collins syndrome (TCS) is an autosomal dominant disorder of craniofacial development caused by mutations in the gene TCOF1. Its gene product, treacle, consists mainly of a central repeat...

The Treacher Collins syndrome (TCOF1) gene product is involved in ribosomal DNA gene transcription by interacting with upstream binding factor

Valdez, Benigno C., Henning, Dale, So, Rolando B., Dixon, Jill, Dixon, Michael J.

Treacher Collins syndrome (TCS) is an autosomal dominant disorder characterized by an abnormality of craniofacial development that arises during early embryogenesis. TCS is caused by mutations in the...

Targeted Deletion of mek5 Causes Early Embryonic Death and Defects in the Extracellular Signal-Regulated Kinase 5/Myocyte Enhancer Factor 2 Cell Survival Pathway

Wang, Xin, Merritt, Anita J., Seyfried, Jan, Guo, Chun, Papadakis, Emmanouil S., Finegan, Katherine G., ...

To elucidate the physiological significance of MEK5 in vivo, we have examined the effect of mek5 gene elimination in mice. Heterozygous mice appear to be healthy and were fertile. However,...

Tcof1/Treacle is required for neural crest cell formation and proliferation deficiencies that cause craniofacial abnormalities

Dixon, Jill, Jones, Natalie C., Sandell, Lisa L., Jayasinghe, Sachintha M., Crane, Jennifer, Rey, Jean-Philippe, ...

Neural crest cells are a migratory cell population that give rise to the majority of the cartilage, bone, connective tissue, and sensory ganglia in the head. Abnormalities in the formation,...

Integration of IRF6 and Jagged2 signalling is essential for controlling palatal adhesion and fusion competence

Richardson, Rebecca J., Dixon, Jill, Jiang, Rulang, Dixon, Michael J.

In mammals, adhesion and fusion of the palatal shelves are essential mechanisms during the development of the secondary palate; failure of these processes leads to the congenital anomaly, cleft...