Kenji Kishi

Characterization of a Membrane-Bound Arginine-Specific Serine Protease from Rat Intestinal Mucosa (2001)

Kishi, Kenji, Yamazaki, Kazuya, Yasuda, Ikuko, Yahagi, Naohisa, Ichinose, Masao, Tsuchiya, Yuichi, ...

Previously we isolated and characterized a membrane-bound, arginine-specific serine protease from pig intestinal mucosa [J. Biol Chem 269, 32985–32991 (1994)]. For further characterization of this...

Clindamycin Suppresses Endotoxin Released by Ceftazidime-Treated Escherichia coli O55:B5 and Subsequent Production of Tumor Necrosis Factor Alpha and Interleukin-1β

Kishi, Kenji, Hirai, Kazuhiro, Hiramatsu, Kazufumi, Yamasaki, Tohru, Nasu, Masaru

Treatment of septicemia caused by Escherichia coli with ceftazidime (CAZ) may be associated with the development of septic shock due to the release of bacterial lipopolysaccharide. We examined the...

Pretreatment of Mice with Clindamycin Improves Survival of Endotoxic Shock by Modulating the Release of Inflammatory Cytokines

Hirata, Norio, Hiramatsu, Kazufumi, Kishi, Kenji, Yamasaki, Tohru, Ichimiya, Tomoku, Nasu, Masaru

Suppression of endotoxin release and subsequent production of inflammatory cytokines is crucial in the treatment of septic shock. We investigated the effect of clindamycin (CLI) on endotoxic shock...

Diversity in Protein Synthesis and Viability of Helicobacter pylori Coccoid Forms in Response to Various Stimuli

Mizoguchi, Hiromoto, Fujioka, Toshio, Kishi, Kenji, Nishizono, Akira, Kodama, Reiji, Nasu, Masaru

The viability of the coccoid forms of Helicobacter pylori was evaluated by assessing protein synthesis. Metabolic labeling studies showed the synthesis of proteins and the specific protein profiles...

Clindamycin Modulates Inflammatory-Cytokine Induction in Lipopolysaccharide-Stimulated Mouse Peritoneal Macrophages

Nakano, Tetsuji, Hiramatsu, Kazufumi, Kishi, Kenji, Hirata, Norio, Kadota, Jun-ichi, Nasu, Masaru

We investigated the mechanism by which clindamycin (CLI) modulates cytokine induction after lipopolysaccharide (LPS) stimulation. Although CLI decreased the intracellular expression levels of tumor...

Clindamycin Suppresses Endotoxin Released by Ceftazidime-Treated Escherichia coli O55:B5 and Subsequent Production of Tumor Necrosis Factor Alpha and Interleukin-1β

Kishi, Kenji, Hirai, Kazuhiro, Hiramatsu, Kazufumi, Yamasaki, Tohru, Nasu, Masaru

Treatment of septicemia caused by Escherichia coli with ceftazidime (CAZ) may be associated with the development of septic shock due to the release of bacterial lipopolysaccharide. We examined the...

Pretreatment of Mice with Clindamycin Improves Survival of Endotoxic Shock by Modulating the Release of Inflammatory Cytokines

Hirata, Norio, Hiramatsu, Kazufumi, Kishi, Kenji, Yamasaki, Tohru, Ichimiya, Tomoku, Nasu, Masaru

Suppression of endotoxin release and subsequent production of inflammatory cytokines is crucial in the treatment of septic shock. We investigated the effect of clindamycin (CLI) on endotoxic shock...

Diversity in Protein Synthesis and Viability of Helicobacter pylori Coccoid Forms in Response to Various Stimuli

Mizoguchi, Hiromoto, Fujioka, Toshio, Kishi, Kenji, Nishizono, Akira, Kodama, Reiji, Nasu, Masaru

The viability of the coccoid forms of Helicobacter pylori was evaluated by assessing protein synthesis. Metabolic labeling studies showed the synthesis of proteins and the specific protein profiles...

Clindamycin Modulates Inflammatory-Cytokine Induction in Lipopolysaccharide-Stimulated Mouse Peritoneal Macrophages

Nakano, Tetsuji, Hiramatsu, Kazufumi, Kishi, Kenji, Hirata, Norio, Kadota, Jun-ichi, Nasu, Masaru

We investigated the mechanism by which clindamycin (CLI) modulates cytokine induction after lipopolysaccharide (LPS) stimulation. Although CLI decreased the intracellular expression levels of tumor...