Investigation on the Welding of High-Strength Aluminum Alloys. (1998)
Birinyi,L. K., Williams,R. D., Voldrich,C. B.
The third progress report is on the investigation on the welding of thick plates of high-strength aluminum alloys. The high-strength alloy plates (24ST, 61SW, 61ST, 75SW, and 75ST) were received in...
Investigation on the Welding of High-Strength Aluminum Alloys. (1998)
Birinyi,L. K., Williams,R. D., Voldrich,C. B.
This report covers tests on welded joints in thick 2S aluminum plate which form a basis for Part I of this investigation (determination of the strength properties of joints in thick, high-strength...
Investigation on the Welding of High-Strength Aluminum Alloys. (1998)
Birinyi,L. K., Williams,R. D., Voldrich,C. B.
Industrial applications of arc- and gas-welded high-strength aluminum alloy assemblies have been limited because of the poor weldability of these alloys and because suitable high-strength filler...
Investigation of the Welding of High-Strength Aluminum Alloys. (1998)
Birinyi,L. K., Williams,R. D., Voldrich,C. B.
This is the fourth report on the investigation on the welding of high-strength aluminum alloys. The three previous reports, have covered in detail preliminary studies on welding techniques for the...
Inducible interleukin-1 gene expression in human vascular smooth muscle cells.
Libby, P, Ordovas, J M, Birinyi, L K, Auger, K R, Dinarello, C A
Interleukin-1 (IL-1) mediates many components of generalized host response to injury and may also contribute to local vascular pathology during immune or inflammatory responses. Because altered...
Inducible interleukin-1 gene expression in human vascular smooth muscle cells.
Libby, P, Ordovas, J M, Birinyi, L K, Auger, K R, Dinarello, C A
Interleukin-1 (IL-1) mediates many components of generalized host response to injury and may also contribute to local vascular pathology during immune or inflammatory responses. Because altered...
Libby, P., Ordovas, J. M., Auger, K. R., Robbins, A. H., Birinyi, L. K., Dinarello, C. A.
Interleukin 1 (IL-1) can induce potentially pathogenic functions of vascular endothelial cells. This mediator was formerly thought to be produced primarily by activated macrophages. We report here...