Lee S. Weinstein

Publication List Details

Period

1995 - 2005

Number

29

Co-Authors

Distinct patterns of abnormal GNAS imprinting in familial and sporadic pseudohypoparathyroidism type IB (2005)

Liu, Jie, Nealon, Julie G., Weinstein, Lee S.

Pseudohypoparathyroidism type IB (PHPIB) is associated with abnormal imprinting of GNAS, the gene encoding the heterotrimeric G protein Gsα and other alternative products. The gene contains three...

Tissue-specific imprinting of the G protein Gs{alpha} is associated with tissue-specific differences in histone methylation (2004)

Sakamoto, Akio, Liu, Jie, Greene, Andrew, Chen, Min, Weinstein, Lee S.

The G protein Gsα is imprinted in a tissue-specific manner, being primarily expressed from the maternal allele in some tissues, such as renal proximal tubules. The Gsα promoter is unmethylated, but...

Distinct patterns of abnormal GNAS imprinting in familial and sporadic pseudohypoparathyroidism type IB (2004)

Liu, Jie, Nealon, Julie G., Weinstein, Lee S.

Pseudohypoparathyroidism type IB (PHPIB) is associated with abnormal imprinting of GNAS, the gene encoding the heterotrimeric G protein Gsα and other alternative products. The gene contains three...

Tissue-specific imprinting of the G protein Gs{alpha} is associated with tissue-specific differences in histone methylation (2004)

Sakamoto, Akio, Liu, Jie, Greene, Andrew, Chen, Min, Weinstein, Lee S.

The G protein Gsα is imprinted in a tissue-specific manner, being primarily expressed from the maternal allele in some tissues, such as renal proximal tubules. The Gsα promoter is unmethylated, but...

Tissue-specific imprinting of the G protein Gs{alpha} is associated with tissue-specific differences in histone methylation (2004)

Sakamoto, Akio, Liu, Jie, Greene, Andrew, Chen, Min, Weinstein, Lee S.

The G protein Gsα is imprinted in a tissue-specific manner, being primarily expressed from the maternal allele in some tissues, such as renal proximal tubules. The Gsα promoter is unmethylated, but...

Distinct patterns of abnormal GNAS imprinting in familial and sporadic pseudohypoparathyroidism type IB (2004)

Liu, Jie, Nealon, Julie G., Weinstein, Lee S.

Pseudohypoparathyroidism type IB (PHPIB) is associated with abnormal imprinting of GNAS, the gene encoding the heterotrimeric G protein Gsα and other alternative products. The gene contains three...

Identification and localization of G protein subunits in human spermatozoa (1999)

Merlet, Françoise, Weinstein, Lee S., Goldsmith, Paul K., Rarick, Tom, Hall, Jerry L., Bisson, Jean-Pierre, ...

Antibodies to α and β subunits of guanine nucleotide regulatory proteins (G proteins) were used to identify which G proteins are present in mature human spermatozoa and to determine their...

A mutation in the heterotrimeric stimulatory guanine nucleotide binding protein α-subunit with impaired receptor-mediated activation because of elevated GTPase activity

Warner, Dennis R., Weinstein, Lee S.

It has been reported that substitution of Arg258, a residue within the GTPase domain of the heterotrimeric guanine nucleotide binding protein (G protein) α-subunit (αs), to alanine (αs-R258A)...

Variable and tissue-specific hormone resistance in heterotrimeric Gs protein α-subunit (Gsα) knockout mice is due to tissue-specific imprinting of the Gsα gene

Yu, Shuhua, Yu, Dawen, Lee, Eric, Eckhaus, Michael, Lee, Randy, Corria, Zakia, ...

Albright hereditary osteodystrophy (AHO), an autosomal dominant disorder characterized by short stature, obesity, and skeletal defects, is associated with heterozygous inactivating mutations of...

Identification of a Methylation Imprint Mark within the Mouse Gnas Locus

Liu, Jie, Yu, Shuhua, Litman, Deborah, Chen, Weiping, Weinstein, Lee S.

The imprinted mouse gene Gnas produces the G protein α-subunit GSα and several other gene products by using alternative promoters and first exons. GSα is maternally expressed in some tissues and...

Paternal versus maternal transmission of a stimulatory G-protein α subunit knockout produces opposite effects on energy metabolism

Yu, Shuhua, Gavrilova, Oksana, Chen, Hui, Lee, Randy, Liu, Jie, Pacak, Karel, ...

Heterozygous disruption of Gnas, the gene encoding the stimulatory G-protein α subunit (Gsα), leads to distinct phenotypes depending on whether the maternal (m–/+) or paternal (+/p–) allele is...

A GNAS1 imprinting defect in pseudohypoparathyroidism type IB

Liu, Jie, Litman, Deborah, Rosenberg, Marjorie J., Yu, Shuhua, Biesecker, Leslie G., Weinstein, Lee S.

Pseudohypoparathyroidism type IB (PHPIB) is characterized by renal resistance to parathyroid hormone (PTH) and the absence of other endocrine or physical abnormalities. Familial PHPIB has been mapped...

Stimulatory G protein directly regulates hypertrophic differentiation of growth plate cartilage in vivo

Bastepe, Murat, Weinstein, Lee S., Ogata, Naoshi, Kawaguchi, Hiroshi, Jüppner, Harald, Kronenberg, Henry M., ...

Stimulatory heterotrimeric G protein (Gs) transduces signals from various cell-surface receptors to adenylyl cyclases, which generate cAMP. The α subunit of Gs (Gsα) is encoded by GNAS (Gnas in...

Identification of the control region for tissue-specific imprinting of the stimulatory G protein α-subunit

Liu, Jie, Chen, Min, Deng, Chuxia, Bourc'his, Déborah, Nealon, Julie G., Erlichman, Beth, ...

Gnas is a complex gene with multiple imprinted promoters. The upstream Nesp and Nespas/Gnasxl promoters are paternally and maternally methylated, respectively. The downstream promoter for the...

Alternative Gnas gene products have opposite effects on glucose and lipid metabolism

Chen, Min, Gavrilova, Oksana, Liu, Jie, Xie, Tao, Deng, Chuxia, Nguyen, Annie T., ...

Gnas is an imprinted gene with multiple gene products resulting from alternative splicing of different first exons onto a common exon 2. These products include stimulatory G protein α-subunit...

Increased glucose tolerance and reduced adiposity in the absence of fasting hypoglycemia in mice with liver-specific Gsα deficiency

Chen, Min, Gavrilova, Oksana, Zhao, Wei-Qin, Nguyen, Annie, Lorenzo, Javier, Shen, Laura, ...

The G protein Gsα is essential for hormone-stimulated cAMP generation and is an important metabolic regulator. We investigated the role of liver Gs-signaling pathways by developing mice with...

A mutation in the heterotrimeric stimulatory guanine nucleotide binding protein α-subunit with impaired receptor-mediated activation because of elevated GTPase activity

Warner, Dennis R., Weinstein, Lee S.

It has been reported that substitution of Arg258, a residue within the GTPase domain of the heterotrimeric guanine nucleotide binding protein (G protein) α-subunit (αs), to alanine (αs-R258A)...

Variable and tissue-specific hormone resistance in heterotrimeric Gs protein α-subunit (Gsα) knockout mice is due to tissue-specific imprinting of the Gsα gene

Yu, Shuhua, Yu, Dawen, Lee, Eric, Eckhaus, Michael, Lee, Randy, Corria, Zakia, ...

Albright hereditary osteodystrophy (AHO), an autosomal dominant disorder characterized by short stature, obesity, and skeletal defects, is associated with heterozygous inactivating mutations of...

Identification of a Methylation Imprint Mark within the Mouse Gnas Locus

Liu, Jie, Yu, Shuhua, Litman, Deborah, Chen, Weiping, Weinstein, Lee S.

The imprinted mouse gene Gnas produces the G protein α-subunit GSα and several other gene products by using alternative promoters and first exons. GSα is maternally expressed in some tissues and...

Paternal versus maternal transmission of a stimulatory G-protein α subunit knockout produces opposite effects on energy metabolism

Yu, Shuhua, Gavrilova, Oksana, Chen, Hui, Lee, Randy, Liu, Jie, Pacak, Karel, ...

Heterozygous disruption of Gnas, the gene encoding the stimulatory G-protein α subunit (Gsα), leads to distinct phenotypes depending on whether the maternal (m–/+) or paternal (+/p–) allele is...

A GNAS1 imprinting defect in pseudohypoparathyroidism type IB

Liu, Jie, Litman, Deborah, Rosenberg, Marjorie J., Yu, Shuhua, Biesecker, Leslie G., Weinstein, Lee S.

Pseudohypoparathyroidism type IB (PHPIB) is characterized by renal resistance to parathyroid hormone (PTH) and the absence of other endocrine or physical abnormalities. Familial PHPIB has been mapped...

Stimulatory G protein directly regulates hypertrophic differentiation of growth plate cartilage in vivo

Bastepe, Murat, Weinstein, Lee S., Ogata, Naoshi, Kawaguchi, Hiroshi, Jüppner, Harald, Kronenberg, Henry M., ...

Stimulatory heterotrimeric G protein (Gs) transduces signals from various cell-surface receptors to adenylyl cyclases, which generate cAMP. The α subunit of Gs (Gsα) is encoded by GNAS (Gnas in...

Identification of the control region for tissue-specific imprinting of the stimulatory G protein α-subunit

Liu, Jie, Chen, Min, Deng, Chuxia, Bourc'his, Déborah, Nealon, Julie G., Erlichman, Beth, ...

Gnas is a complex gene with multiple imprinted promoters. The upstream Nesp and Nespas/Gnasxl promoters are paternally and maternally methylated, respectively. The downstream promoter for the...

Alternative Gnas gene products have opposite effects on glucose and lipid metabolism

Chen, Min, Gavrilova, Oksana, Liu, Jie, Xie, Tao, Deng, Chuxia, Nguyen, Annie T., ...

Gnas is an imprinted gene with multiple gene products resulting from alternative splicing of different first exons onto a common exon 2. These products include stimulatory G protein α-subunit...

Increased glucose tolerance and reduced adiposity in the absence of fasting hypoglycemia in mice with liver-specific Gsα deficiency

Chen, Min, Gavrilova, Oksana, Zhao, Wei-Qin, Nguyen, Annie, Lorenzo, Javier, Shen, Laura, ...

The G protein Gsα is essential for hormone-stimulated cAMP generation and is an important metabolic regulator. We investigated the role of liver Gs-signaling pathways by developing mice with...

β cell-specific deficiency of the stimulatory G protein α-subunit Gsα leads to reduced β cell mass and insulin-deficient diabetes

Xie, Tao, Chen, Min, Zhang, Qing-Hong, Ma, Zheng, Weinstein, Lee S.

The G protein α-subunit Gsα is required for hormone-stimulated cAMP generation. In pancreatic β cells, Gsα mediates the signaling of glucagon-like peptide 1 and other incretin hormones, which are...

Osteoblastic regulation of B lymphopoiesis is mediated by Gsα-dependent signaling pathways

Wu, Joy Y., Purton, Louise E., Rodda, Stephen J., Chen, Min, Weinstein, Lee S., McMahon, Andrew P., ...

Osteoblasts play an increasingly recognized role in supporting hematopoietic development and recently have been implicated in the regulation of B lymphopoiesis. Here we demonstrate that the...

Severe Obesity and Insulin Resistance due to Deletion of the Maternal Gsα Allele Is Reversed by Paternal Deletion of the Gsα Imprint Control Region

Xie, Tao, Chen, Min, Gavrilova, Oksana, Lai, Edwin W., Liu, Jie, Weinstein, Lee S.

The G protein α-subunit Gsα mediates receptor-stimulated cAMP production and is imprinted with reduced expression from the paternal allele in specific tissues. Disruption of the Gsα maternal (but...