Michelle Olive

Publication List Details

Period

1998 - 1998

Number

16

Co-Authors

hnRNP A1 Recruited to an Exon In Vivo Can Function as an Exon Splicing Silencer

Olive, Michelle, Gesnel, Marie-Claude, Breathnach, Richard

Some exons contain exon splicing silencers. Their activity is frequently balanced by that of splicing enhancers, and this is important to ensure correct relative levels of alternatively spliced...

Tumor Necrosis Factor Alpha Inhibits Type I Collagen Synthesis through Repressive CCAAT/Enhancer-Binding Proteins

Greenwel, Patricia, Tanaka, Shizuko, Penkov, Dmitri, Zhang, Wen, Olive, Michelle, Moll, Jonathan, ...

Extracellular matrix (ECM) formation and remodeling are critical processes for proper morphogenesis, organogenesis, and tissue repair. The proinflammatory cytokine tumor necrosis factor alpha...

A Dominant-Negative Inhibitor of CREB Reveals that It Is a General Mediator of Stimulus-Dependent Transcription of c-fos

Ahn, Sohyun, Olive, Michelle, Aggarwal, Seema, Krylov, Dmitry, Ginty, David D., Vinson, Charles

Several studies have characterized the upstream regulatory region of c-fos, and identified cis-acting elements termed the cyclic AMP (cAMP) response elements (CREs) that are critical for c-fos...

Life without white fat: a transgenic mouse

Moitra, Jaideep, Mason, Mark M., Olive, Michelle, Krylov, Dmitry, Gavrilova, Oksana, Marcus-Samuels, Bernice, ...

We have generated a transgenic mouse with no white fat tissue throughout life. These mice express a dominant-negative protein, termed A-ZIP/F, under the control of the adipose-specific aP2...

Bone marrow–derived immune cells regulate vascular disease through a p27Kip1-dependent mechanism

Boehm, Manfred, Olive, Michelle, True, Andrea L., Crook, Martin F., San, Hong, Qu, Xuan, ...

The cyclin-dependent kinase inhibitors are key regulators of cell cycle progression. Although implicated in carcinogenesis, they inhibit the proliferation of a variety of normal cell types, and their...

Progressive vascular smooth muscle cell defects in a mouse model of Hutchinson–Gilford progeria syndrome

Varga, Renee, Eriksson, Maria, Erdos, Michael R., Olive, Michelle, Harten, Ingrid, Kolodgie, Frank, ...

Children with Hutchinson–Gilford progeria syndrome (HGPS) suffer from dramatic acceleration of some symptoms associated with normal aging, most notably cardiovascular disease that eventually leads...

hnRNP A1 Recruited to an Exon In Vivo Can Function as an Exon Splicing Silencer

Olive, Michelle, Gesnel, Marie-Claude, Breathnach, Richard

Some exons contain exon splicing silencers. Their activity is frequently balanced by that of splicing enhancers, and this is important to ensure correct relative levels of alternatively spliced...

Tumor Necrosis Factor Alpha Inhibits Type I Collagen Synthesis through Repressive CCAAT/Enhancer-Binding Proteins

Greenwel, Patricia, Tanaka, Shizuko, Penkov, Dmitri, Zhang, Wen, Olive, Michelle, Moll, Jonathan, ...

Extracellular matrix (ECM) formation and remodeling are critical processes for proper morphogenesis, organogenesis, and tissue repair. The proinflammatory cytokine tumor necrosis factor alpha...

A Dominant-Negative Inhibitor of CREB Reveals that It Is a General Mediator of Stimulus-Dependent Transcription of c-fos

Ahn, Sohyun, Olive, Michelle, Aggarwal, Seema, Krylov, Dmitry, Ginty, David D., Vinson, Charles

Several studies have characterized the upstream regulatory region of c-fos, and identified cis-acting elements termed the cyclic AMP (cAMP) response elements (CREs) that are critical for c-fos...

Life without white fat: a transgenic mouse

Moitra, Jaideep, Mason, Mark M., Olive, Michelle, Krylov, Dmitry, Gavrilova, Oksana, Marcus-Samuels, Bernice, ...

We have generated a transgenic mouse with no white fat tissue throughout life. These mice express a dominant-negative protein, termed A-ZIP/F, under the control of the adipose-specific aP2...

Bone marrow–derived immune cells regulate vascular disease through a p27Kip1-dependent mechanism

Boehm, Manfred, Olive, Michelle, True, Andrea L., Crook, Martin F., San, Hong, Qu, Xuan, ...

The cyclin-dependent kinase inhibitors are key regulators of cell cycle progression. Although implicated in carcinogenesis, they inhibit the proliferation of a variety of normal cell types, and their...

Progressive vascular smooth muscle cell defects in a mouse model of Hutchinson–Gilford progeria syndrome

Varga, Renee, Eriksson, Maria, Erdos, Michael R., Olive, Michelle, Harten, Ingrid, Kolodgie, Frank, ...

Children with Hutchinson–Gilford progeria syndrome (HGPS) suffer from dramatic acceleration of some symptoms associated with normal aging, most notably cardiovascular disease that eventually leads...

p21Cip1 modulates arterial wound repair through the stromal cell–derived factor-1/CXCR4 axis in mice

Olive, Michelle, Mellad, Jason A., Beltran, Leilani E., Ma, Mingchao, Cimato, Thomas, Noguchi, Audrey C., ...

Cyclin-dependent kinase inhibitors, including p21Cip1, are implicated in cell turnover and are active players in cardiovascular wound repair. Here, we show that p21Cip1 orchestrates the complex...

KIS protects against adverse vascular remodeling by opposing stathmin-mediated VSMC migration in mice

Langenickel, Thomas H., Olive, Michelle, Boehm, Manfred, San, Hong, Crook, Martin F., Nabel, Elizabeth G.

Vascular proliferative diseases are characterized by VSMC proliferation and migration. Kinase interacting with stathmin (KIS) targets 2 key regulators of cell proliferation and migration, the...

A farnesyltransferase inhibitor prevents both the onset and late progression of cardiovascular disease in a progeria mouse model

Capell, Brian C., Olive, Michelle, Erdos, Michael R., Cao, Kan, Faddah, Dina A., Tavarez, Urraca L., ...

Hutchinson-Gilford progeria syndrome (HGPS) is the most dramatic form of human premature aging. Death occurs at a mean age of 13 years, usually from heart attack or stroke. Almost all cases of HGPS...