Outi Kopra

Publication List Details

Period

2001 - 2008

Number

17

Co-Authors

Brain gene expression profiles of Cln1and Cln5deficient mice unravels common molecular pathways underlying neuronal degeneration in NCL diseases (2008)

Von Schantz, Carina, Saharinen, Juha, Kopra, Outi, Cooper, Jonathan D, Gentile, Massimiliano, Hovatta, Iiris, ...

Abstract Background The neuronal ceroid lipofuscinoses (NCL) are a group of children's inherited neurodegenerative disorders, characterized by blindness, early dementia and pronounced cortical...

Deficiency of the INCL protein Ppt1 results in changes in ectopic F1-ATP synthase and altered cholesterol metabolism (2008)

Lyly, Annina, Marjavaara, Sanna K., Kyttälä, Aija, Uusi-Rauva, Kristiina, Luiro, Kaisu, Kopra, Outi, ...

Infantile neuronal ceroid lipofuscinosis (INCL) is a severe neurodegenerative disease caused by deficiency of palmitoyl protein thioesterase 1 (PPT1). INCL results in dramatic loss of thalamocortical...

Glycosylation, transport, and complex formation of palmitoyl protein thioesterase 1 (PPT1) – distinct characteristics in neurons (2007)

Lyly, Annina, Von Schantz, Carina, Salonen, Tarja, Kopra, Outi, Saarela, Jani, Jauhiainen, Matti, ...

Abstract Background Neuronal ceroid lipofuscinoses (NCLs) are collectively the most common type of recessively inherited childhood encephalopathies. The most severe form of NCL, infantile neuronal...

The neuronal ceroid lipofuscinosis Cln8gene expression is developmentally regulated in mouse brain and up-regulated in the hippocampal kindling model of epilepsy (2005)

Lonka, Liina, Aalto, Antti, Kopra, Outi, Kuronen, Mervi, Kokaia, Zaal, Saarma, Mart, ...

Abstract Background The neuronal ceroid lipofuscinoses (NCLs) are a group of inherited neurodegenerative disorders characterized by accumulation of autofluorescent material in many tissues,...

Hydrolethalus syndrome is caused by a missense mutation in a novel gene HYLS1 (2005)

Mee, Lisa, Honkala, Heli, Kopra, Outi, Vesa, Jouni, Finnilä, Saara, Visapää, Ilona, ...

Hydrolethalus syndrome (HLS) is an autosomal recessive lethal malformation syndrome characterized by multiple developmental defects of fetus. We have earlier mapped and restricted the HLS region to a...

Hydrolethalus syndrome is caused by a missense mutation in a novel gene HYLS1 (2005)

Mee, Lisa, Honkala, Heli, Kopra, Outi, Vesa, Jouni, Finnilä, Saara, Visapää, Ilona, ...

Hydrolethalus syndrome (HLS) is an autosomal recessive lethal malformation syndrome characterized by multiple developmental defects of fetus. We have earlier mapped and restricted the HLS region to a...

A Mouse Model for Finnish Variant Late Infantile Neuronal Ceroid Lipofuscinosis, CLN5, reveals neuropathology associated with early aging (2004)

Kopra, Outi, Vesa, Jouni, Von Schantz, Carina, Manninen, Tuula, Minye, Helena, Fabritius, Anna-Liisa, ...

Neuronal ceroid lipofuscinoses (NCL) comprise the most common group of childhood encephalopathies caused by mutations in eight genetic loci, CLN1-CLN8. Here we have developed a novel mouse model for...

A mouse model for Finnish variant late infantile neuronal ceroid lipofuscinosis, CLN5, reveals neuropathology associated with early aging (2004)

Kopra, Outi, Vesa, Jouni, Von Schantz, Carina, Manninen, Tuula, Minye, Helena, Fabritius, Anna-Liisa, ...

Neuronal ceroid lipofuscinoses (NCL) comprise the most common group of childhood encephalopathies caused by mutations in eight genetic loci, CLN1–CLN8. Here, we have developed a novel mouse model...

A Mouse Model for Finnish Variant Late Infantile Neuronal Ceroid Lipofuscinosis, CLN5, reveals neuropathology associated with early aging (2004)

Kopra, Outi, Vesa, Jouni, Von Schantz, Carina, Manninen, Tuula, Minye, Helena, Fabritius, Anna-Liisa, ...

Neuronal ceroid lipofuscinoses (NCL) comprise the most common group of childhood encephalopathies caused by mutations in eight genetic loci, CLN1-CLN8. Here we have developed a novel mouse model for...

MOLECULAR DEFECT OF THE RAPADILINO SYNDROME EXPANDS THE PHENOTYPE SPECTRUM OF RECQL DISEASES (2003)

Siitonen, H. Annika, Kopra, Outi, Kääriäinen, Helena, Haravuori, Henna, Winter, Robin M., Säämänen, Anna-Marja, ...

The RECQL4 helicase gene is a member of the RECQL gene family, mutated in some Rothmund-Thomson syndrome (RTS) patients. Other members of this gene family are BLM mutated in Bloom syndrome, WRN...

Molecular defect of RAPADILINO syndrome expands the phenotype spectrum of RECQL diseases (2003)

Siitonen, H. Annika, Kopra, Outi, Kääriäinen, Helena, Haravuori, Henna, Winter, Robin M., Säämänen, Anna-Marja, ...

The RECQL4 helicase gene is a member of the RECQL gene family, mutated in some Rothmund–Thomson syndrome (RTS) patients. Other members of this gene family are BLM mutated in Bloom syndrome, WRN...

MOLECULAR DEFECT OF THE RAPADILINO SYNDROME EXPANDS THE PHENOTYPE SPECTRUM OF RECQL DISEASES (2003)

Siitonen, H. Annika, Kopra, Outi, Kääriäinen, Helena, Haravuori, Henna, Winter, Robin M., Säämänen, Anna-Marja, ...

The RECQL4 helicase gene is a member of the RECQL gene family, mutated in some Rothmund-Thomson syndrome (RTS) patients. Other members of this gene family are BLM mutated in Bloom syndrome, WRN...

CLN3 protein is targeted to neuronal synapses but excluded from synaptic vesicles: new clues to Batten disease (2001)

Luiro, Kaisu, Kopra, Outi, Lehtovirta, Maarit, Jalanko, Anu

Batten disease (juvenile neuronal ceroid lipofuscinosis, JNCL), the most common neurodegenerative disease of childhood, is caused by mutations in the CLN3 gene encoding a putative transmembrane...