Patrick Leahy

Supplemental Tables: 6 Correspondence: (2008)

Georgiana Cheng, Anita P. Merriam, Bendi Gong, Patrick Leahy, Sangeeta Khanna, John D. Porter, ...

and muscle group-specific gene expression patterns shape postnatal development of the novel extraocular muscle phenotype

p90 ribosomal S6 kinase 2 exerts a tonic brake on G protein-coupled receptor signaling. (2006)

Sheffler, Douglas J., Kroeze, Wesley K., Garcia, Bonnie G., Deutch, Ariel Y., Hufeisen, Sandra J., Leahy, Patrick, ...

G protein-coupled receptors (GPCRs) are essential for normal central CNS function and represent the proximal site(s) of action for most neurotransmitters and many therapeutic drugs, including typical...

Temporal gene expression profiling of dystrophin-deficient (mdx) mouse diaphragm identifies conserved and muscle group-specific mechanisms in the pathogenesis of muscular dystrophy (2004)

Porter, John D., Merriam, Anita P., Leahy, Patrick, Gong, Bendi, Feuerman, Jason, Cheng, Georgiana, ...

Mutations in dystrophin are the proximate cause of Duchenne muscular dystrophy (DMD), but pathogenic mechanisms linking the absence of dystrophin from the sarcolemma to myofiber necrosis are not...

Dissection of temporal gene expression signatures of affected and spared muscle groups in dystrophin-deficient (mdx) mice (2003)

Porter, John D., Merriam, Anita P., Leahy, Patrick, Gong, Bendi, Khanna, Sangeeta

Although dystrophin mutations are the proximate cause of Duchenne muscular dystrophy (DMD), interactions among heterogeneous downstream mechanisms may be key phenotypic determinants. Temporal gene...

Temporal gene expression profiling of dystrophin-deficient (mdx) mouse diaphragm identifies conserved and muscle group-specific mechanisms in the pathogenesis of muscular dystrophy (2003)

Porter, John D., Merriam, Anita P., Leahy, Patrick, Gong, Bendi, Feuerman, Jason, Cheng, Georgiana, ...

Mutations in dystrophin are the proximate cause of Duchenne muscular dystrophy (DMD), but pathogenic mechanisms linking the absence of dystrophin from the sarcolemma to myofiber necrosis are not...

Temporal gene expression profiling of dystrophin-deficient (mdx) mouse diaphragm identifies conserved and muscle group-specific mechanisms in the pathogenesis of muscular dystrophy (2003)

Porter, John D., Merriam, Anita P., Leahy, Patrick, Gong, Bendi, Feuerman, Jason, Cheng, Georgiana, ...

Mutations in dystrophin are the proximate cause of Duchenne muscular dystrophy (DMD), but pathogenic mechanisms linking the absence of dystrophin from the sarcolemma to myofiber necrosis are not...

A chronic inflammatory response dominates the skeletal muscle molecular signature in dystrophin-deficient mdx mice (2002)

Porter, John D., Khanna, Sangeeta, Kaminski, Henry J., Rao, J. Sunil, Merriam, Anita P., Richmonds, Chelliah R., ...

Mutations in dystrophin cause Duchenne muscular dystrophy (DMD), but absent dystrophin does not invariably cause necrosis in all muscles, life stages and species. Using DNA microarray, we established...

The Linear-Size Evolution of Classical Double Radio Sources (1995)

Neeser, Mark J., Eales, Stephen A., Law-Green, J. Duncan, Leahy, Patrick, Rawlings, Steve

Recent investigations of how the median size of extragalactic radio sources change with redshift have produced inconsistent results. Eales compared the radio and optical properties of a bright 3C and...

Investigation of the flow of fluids past solid bodies. (1942)

Leahy, Patrick.

Thesis (Ph. D.)--University College Dublin, 1942.

Investigation of the flow of fluids past solid bodies. (1942)

Leahy, Patrick.

Thesis (Ph. D.)--University College Dublin, 1942.

p90 ribosomal S6 kinase 2 exerts a tonic brake on G protein-coupled receptor signaling

Sheffler, Douglas J., Kroeze, Wesley K., Garcia, Bonnie G., Deutch, Ariel Y., Hufeisen, Sandra J., Leahy, Patrick, ...

G protein-coupled receptors (GPCRs) are essential for normal central CNS function and represent the proximal site(s) of action for most neurotransmitters and many therapeutic drugs, including typical...

p90 ribosomal S6 kinase 2 exerts a tonic brake on G protein-coupled receptor signaling

Sheffler, Douglas J., Kroeze, Wesley K., Garcia, Bonnie G., Deutch, Ariel Y., Hufeisen, Sandra J., Leahy, Patrick, ...

G protein-coupled receptors (GPCRs) are essential for normal central CNS function and represent the proximal site(s) of action for most neurotransmitters and many therapeutic drugs, including typical...