S. Kostin

Publication List Details

Period

2002 - 2008

Number

65

Co-Authors

Expression profiling of cardiac genes in Tako-Tsubo cardiomyopathy: insight into a new cardiac entity (2008)

Nef, H. M., Mollmann, H., Troidl, C., Kostin, S., Bottger, T., Voss, S., ...

Tako-Tsubo cardiomyopathy (TTC) is characterized by a transient contractile dysfunction, but its specific pathomechanism remains unknown. Thus, we performed a systematic expression profiling of genes...

Atrial extracellular matrix remodeling in patients with atrial fibrillation (2008)

Polyakova, V., Miyagawa, S., Szalay, Z., Risteli, J., Kostin, S.

Background: Atrial fibrillation (AF) is the most frequent clinical arrhythmia. Atrial fibrosis is an important factor in initiating and maintaining AF. However, the collagen turnover and its...

Sirt7 Increases Stress Resistance of Cardiomyocytes and Prevents Apoptosis and Inflammatory Cardiomyopathy in Mice (2008)

Vakhrusheva, O., Smolka, C., Gajawada, P., Kostin, S., Boettger, T., Kubin, T., ...

Sirt7 is a member of the mammalian sirtuin family consisting of 7 genes, Sirt1 to Sirt7, which all share a homology to the founding family member, the yeast Sir2 gene. Most sirtuins are supposed to...

Cellular cardiomyoplasty: improvement of left ventricular function correlates with the release of cardioactive cytokines (2007)

Ebelt, H., Jungblut, M., Zhang, Y., Kubin, T., Kostin, S., Technau, A., ...

A growing number of studies are reporting beneficial effects of the transplantation of alleged cardiac stem cells into diseased hearts after myocardial infarction. However, the mechanisms by which...

Cellular cardiomyoplasty: improvement of left ventricular function correlates with the release of cardioactive cytokines (2007)

Ebelt, H., Jungblut, M., Zhang, Y., Kubin, T., Kostin, S., Technau, A., ...

A growing number of studies are reporting beneficial effects of the transplantation of alleged cardiac stem cells into diseased hearts after myocardial infarction. However, the mechanisms by which...

Endothelial nitric oxide synthase activity is essential for vasodilation during blood flow recovery but not for arteriogenesis (2007)

Mees, B., Wagner, S., Ninci, E., Tribulova, S., Martin, S., Van Haperen, R., ...

OBJECTIVE: Arteriogenesis is the major mechanism of vascular growth, which is able to compensate for blood flow deficiency after arterial occlusion. Endothelial nitric oxide synthase (eNOS) activity...

Extracellular RNA mediates endothelial-cell permeability via vascular endothelial growth factor (2007)

Fischer, S., Gerriets, T., Wessels, C., Walberer, M., Kostin, S., Stolz, E., ...

Cell injury leads to exposure of intracellular material and is associated with increased permeability of vessels in the vicinity of the damage. Here, we demonstrate that natural extracellular RNA as...

Sympathoadrenergic overstimulation in Tako-Tsubo cardiomyopathy triggered by physical and emotional stress (2007)

Nef, H. M., Mollmann, H., Hilpert, P., Masseli, F., Kostin, S., Troidl, C., ...

Tako-Tsubo cardiomyopathy (TTC) which is usually precipitated by profound emotional stress has been widely reported in the past. Recently, several co-morbidities have been found to be associated with...

Tako-Tsubo cardiomyopathy: intraindividual structural analysis in the acute phase and after functional recovery (2007)

Nef, H. M., Mollmann, H., Kostin, S., Troidl, C., Voss, S., Weber, M., ...

AIMS: To gain more insight into the phenomenon of Tako-Tsubo cardiomyopathy (TTC), the purpose of the present study was to investigate the myocardial structure in the acute phase of TTC and after...

S100A4 is upregulated in injured myocardium and promotes growth and survival of cardiac myocytes (2007)

Schneider, M., Kostin, S., Strom, C. C., Aplin, M., Lyngbaek, S., Theilade, J., ...

OBJECTIVE: The multifunctional Ca2+-binding protein S100A4 (also known as Mts1 and Fsp1) is involved in fibrosis and tissue remodeling in several diseases including cancer, kidney fibrosis, central...

Ischemic acidosis causes apoptosis in coronary endothelial cells through activation of caspase-12 (2007)

Kumar, S., Kasseckert, S., Kostin, S., Abdallah, Y., Schafer, C., Kaminski, A., ...

OBJECTIVE: Myocardial ischemia has been shown to induce apoptosis of endothelial cells (EC). However, the mechanism of this endothelial injury is still poorly understood. To analyse the signaling...

Reduced sarcoplasmic reticulum Ca2+ -ATPase activity and dephosphorylated phospholamban contribute to contractile dysfunction in human hibernating myocardium (2006)

Nef, H. M., Mollmann, H., Skwara, W., Bolck, B., Schwinger, R. H., Hamm, Ch, ...

Human hibernating myocardium (HHM) is characterized by reversible contractile dysfunction during chronic ischemia. A disturbed calcium-homeostasis is a decisive factor for reduced functional capacity...

Reduced sarcoplasmic reticulum Ca2+ -ATPase activity and dephosphorylated phospholamban contribute to contractile dysfunction in human hibernating myocardium (2006)

Nef, H. M., Mollmann, H., Skwara, W., Bolck, B., Schwinger, R. H., Hamm, Ch, ...

Human hibernating myocardium (HHM) is characterized by reversible contractile dysfunction during chronic ischemia. A disturbed calcium-homeostasis is a decisive factor for reduced functional capacity...

Bone marrow-derived cells contribute to infarct remodelling (2006)

Mollmann, H., Nef, H. M., Kostin, S., Von Kalle, C., Pilz, I., Weber, M., ...

OBJECTIVE: The paradigm that cardiac myocytes are non-proliferating and terminally differentiated cells has recently been challenged by several studies reporting the ability of bone marrow-derived...

Reduced sarcoplasmic reticulum Ca2+ -ATPase activity and dephosphorylated phospholamban contribute to contractile dysfunction in human hibernating myocardium (2006)

Nef, H. M., Mollmann, H., Skwara, W., Bolck, B., Schwinger, R. H., Hamm, Ch, ...

Human hibernating myocardium (HHM) is characterized by reversible contractile dysfunction during chronic ischemia. A disturbed calcium-homeostasis is a decisive factor for reduced functional capacity...

Ischemia depletes dystrophin and inhibits protein synthesis in the canine heart: mechanisms of myocardial ischemic injury (2005)

Rodriguez, M., Cai, W. J., Kostin, S., Lucchesi, B. R., Schaper, J.

In this study we tested our previous hypothesis that ischemia is a multifactorial injurious event involving all components of the myocyte simultaneously. This hypothesis was based on ultrastructural...

Pathways of myocyte death: implications for development of clinical laboratory biomarkers (2005)

Kostin, S.

The recognition that cardiac myocytes die by multiple mechanisms and thus substantially affect ventricular remodeling in diseased human hearts supports the concept of ongoing myocyte death in the...

Carvedilol improves myocardial contractility compared with metoprolol in patients with chronic hibernating myocardium after revascularization (2005)

Schwarz, E. R., Gupta, R., Diep, T. P., Nowak, B., Kostin, S., Grohmann, B., ...

BACKGROUND: We tested the hypothesis of whether carvedilol delays morphologic degeneration and improves functional outcome compared with metoprolol tartrate in patients with hibernating myocardium...

Gap junctional remodeling by hypoxia in cultured neonatal rat ventricular myocytes (2005)

Zeevi-Levin, N., Barac, Y. D., Reisner, Y., Reiter, I., Yaniv, G., Meiry, G., ...

OBJECTIVES: Altered gap junctional coupling of ventricular myocytes plays an important role in arrhythmogenesis in ischemic heart disease. Since hypoxia is a major component of ischemia, we tested...

Transforming growth factor-beta1 downregulates beating frequency and remodeling of cultured rat adult cardiomyocytes (2005)

Kubin, T., Tomars, M., Fach, C., Hein, S., Bramlage, P., Shim, G. J., ...

We have observed increased levels of transforming growth factor-beta1 (TGF-beta1) in human hibernating myocardium (HM). Impaired ventricular function in HM is known to be restored to normal following...

Downregulation of apoptosis-inducing factor in harlequin mutant mice sensitizes the myocardium to oxidative stress-related cell death and pressure overload-induced decompensation (2005)

Van Empel, V. P., Bertrand, A. T., Van Der Nagel, R., Kostin, S., Doevendans, P. A., Crijns, H. J., ...

Apoptosis-inducing factor (AIF), or programmed cell death 8 (Pdcd8), is a highly conserved, ubiquitous flavoprotein localized in the mitochondrial intermembrane space. In vivo, AIF provides...

Downregulation of apoptosis-inducing factor in harlequin mutant mice sensitizes the myocardium to oxidative stress-related cell death and pressure overload-induced decompensation (2005)

Van Empel, V. P., Bertrand, A. T., Van Der Nagel, R., Kostin, S., Doevendans, P. A., Crijns, H. J., ...

Apoptosis-inducing factor (AIF), or programmed cell death 8 (Pdcd8), is a highly conserved, ubiquitous flavoprotein localized in the mitochondrial intermembrane space. In vivo, AIF provides...

Transforming growth factor-beta1 downregulates beating frequency and remodeling of cultured rat adult cardiomyocytes (2005)

Kubin, T., Tomars, M., Fach, C., Hein, S., Bramlage, P., Shim, G. J., ...

We have observed increased levels of transforming growth factor-beta1 (TGF-beta1) in human hibernating myocardium (HM). Impaired ventricular function in HM is known to be restored to normal following...

Gap junctional remodeling by hypoxia in cultured neonatal rat ventricular myocytes (2005)

Zeevi-Levin, N., Barac, Y. D., Reisner, Y., Reiter, I., Yaniv, G., Meiry, G., ...

OBJECTIVES: Altered gap junctional coupling of ventricular myocytes plays an important role in arrhythmogenesis in ischemic heart disease. Since hypoxia is a major component of ischemia, we tested...

Carvedilol improves myocardial contractility compared with metoprolol in patients with chronic hibernating myocardium after revascularization (2005)

Schwarz, E. R., Gupta, R., Diep, T. P., Nowak, B., Kostin, S., Grohmann, B., ...

BACKGROUND: We tested the hypothesis of whether carvedilol delays morphologic degeneration and improves functional outcome compared with metoprolol tartrate in patients with hibernating myocardium...

Pathways of myocyte death: implications for development of clinical laboratory biomarkers (2005)

Kostin, S.

The recognition that cardiac myocytes die by multiple mechanisms and thus substantially affect ventricular remodeling in diseased human hearts supports the concept of ongoing myocyte death in the...

Ischemia depletes dystrophin and inhibits protein synthesis in the canine heart: mechanisms of myocardial ischemic injury (2005)

Rodriguez, M., Cai, W. J., Kostin, S., Lucchesi, B. R., Schaper, J.

In this study we tested our previous hypothesis that ischemia is a multifactorial injurious event involving all components of the myocyte simultaneously. This hypothesis was based on ultrastructural...

Bone marrow-derived cells do not incorporate into the adult growing vasculature (2004)

Ziegelhoeffer, T., Fernandez, B., Kostin, S., Heil, M., Voswinckel, R., Helisch, A., ...

Bone marrow-Derived cells have been proposed to form new vessels or at least incorporate into growing vessels in adult organisms under certain physiological and pathological conditions. We...

Matrix metalloproteinases and their tissue inhibitors in pressure-overloaded human myocardium during heart failure progression (2004)

Polyakova, V., Hein, S., Kostin, S., Ziegelhoeffer, T., Schaper, J.

OBJECTIVES: We studied the role of matrix metalloproteinases (MMPs) and their tissue inhibitors (TIMPs) in fibrosis formation in the transition from hypertrophy to heart failure (HF) as well as the...

Remodeling of the adventitia during coronary arteriogenesis (2003)

Cai, W. J., Koltai, S., Kocsis, E., Scholz, D., Kostin, S., Luo, X., ...

We studied the role of the adventitia in adaptive arteriogenesis during the phase of active growth of coronary collateral vessels (CV) induced by chronic occlusion of the left circumflex coronary...

Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms (2003)

Hein, S., Arnon, E., Kostin, S., Schonburg, M., Elsasser, A., Polyakova, V., ...

BACKGROUND: The progression of compensated hypertrophy to heart failure (HF) is still debated. We investigated patients with isolated valvular aortic stenosis and differing degrees of left...

Myocytes die by multiple mechanisms in failing human hearts (2003)

Kostin, S., Pool, L., Elsasser, A., Hein, S., Drexler, H. C., Arnon, E., ...

We tested the hypothesis that myocyte loss in failing human hearts occurs by different mechanisms: apoptosis, oncosis, and autophagic cell death. Explanted hearts from 19 patients with idiopathic...

Gap junction remodeling and altered connexin43 expression in the failing human heart (2003)

Kostin, S., Rieger, M., Dammer, S., Hein, S., Richter, M., Klovekorn, W. P., ...

Gap junctions (GJ) are important determinants of cardiac conduction and the evidence has recently emerged that altered distribution of these junctions and changes in the expression of their...

Blood monocyte concentration is critical for enhancement of collateral artery growth (2002)

Heil, M., Ziegelhoeffer, T., Pipp, F., Kostin, S., Martin, S., Clauss, M., ...

0.1152/ajpheart. 01098.2001.-Arteriogenesis has been associated with the presence of monocytes/macrophages within the collateral vessel wall. We tested the hypothesis that arteriogenesis is...

Zonula occludens-1 and connexin 43 expression in the failing human heart

Kostin, S.

Focal disorganization of gap junctional distribution and down-regulation of the major gap junctional protein connexin 43 are typical features of myocardial remodelling in the failing human heart....

Importance of bicarbonate transport for ischaemia-induced apoptosis of coronary endothelial cells

Kumar, S., Kasseckert, S., Kostin, S., Abdallah, Y., Piper, H. M., Steinhoff, G., ...

Bicarbonate transport (BT) has been previously shown to participate in apoptosis induced by various stress factors. However, the precise role of BT in ischaemia-induced apoptosis is still unknown. To...