S. S. Sommer

Publication List Details

Period

1989 - 2008

Number

85

Co-Authors

PAP-LMPCR for improved, allele-specific footprinting and automated chromatin fine structure analysis (2008)

Ingram, R., Gao, C., LeBon, J., Liu, Q., Mayoral, R. J., Sommer, S. S., ...

The analysis of chromatin fine structure and transcription factor occupancy of differentially expressed genes by in vivo footprinting and ligation-mediated-PCR (LMPCR) is a powerful tool to...

Haemophilia B: database of point mutations and short additions and deletions---eighth edition (1997)

Giannelli Green, P. M. Green, S. S. Sommer, M. Ludwig, R. Schwaab, ...

The eighth edition of the haemophilia B database (http://www.umds.ac.uk/molgen/haemBdatabase.htm ) lists in an easily accessible form all known factor IX mutations due to small changes (base...

Haemophilia B: database of point mutations and short additions and deletions, 7th edition (1996)

F. Giannelli, P. M. Green, S. S. Sommer, M-c. Poon, M. Ludwig, R. Schwaab, ...

The seventh edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of <30...

Factor VIII gene inversions in severe hemophilia A: results of an international consortium study (1995)

Antonarakis, S E, Rossiter, J P, Young, M, Horst, J, DE MOERLOOSE, P; U0022326, Sommer, S S, ...

Twenty-two molecular diagnostic laboratories from 14 countries participated in a consortium study to estimate the impact of Factor VIII gene inversions in severe hemophilia A. A total of 2,093...

Haemophilia B (sixth edition): a database of point mutations and short additions and deletions (1995)

F. Giannelli, P. M. Green, S. S. Sommer, M. Ludwig, R. Schwaab, ...

The sixth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of <30...

Haemophilia B: database of point mutations and short additions and deletions, fifth edition, 1994 (1994)

Giannelli, F., Green, P.M., Sommer, S.S., Lillicrap, D.P., Ludwig, M., Schwaab, R., ...

The fifth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of

Mutation detection by highly sensitive methods indicates that p53 gene mutations in breast cancer can have important prognostic value.

Kovach, J S, Hartmann, A, Blaszyk, H, Cunningham, J, Schaid, D, Sommer, S S

Human cancer cells with a mutated p53 tumor-suppressor gene have a selective growth advantage and may exhibit resistance to ionizing radiation and certain chemotherapeutic agents. To examine the...

Haemophilia B (sixth edition): a database of point mutations and short additions and deletions.

Giannelli, F, Green, P M, Sommer, S S, Poon, M C, Ludwig, M, Schwaab, R, ...

The sixth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of

Haemophilia B: database of point mutations and short additions and deletions, 7th edition.

Giannelli, F, Green, P M, Sommer, S S, Poon, M C, Ludwig, M, Schwaab, R, ...

The seventh edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of

Haemophilia B: database of point mutations and short additions and deletions--eighth edition.

Giannelli, F, Green, P M, Sommer, S S, Poon, M, Ludwig, M, Schwaab, R, ...

The eighth edition of the haemophilia B database (http://www.umds.ac. uk/molgen/haemBdatabase.htm ) lists in an easily accessible form all known factor IX mutations due to small changes (base...

Co-curing of plasmids affecting killer double-stranded RNAs of Saccharomyces cerevisiae: [HOK], [NEX], and the abundance of L are related and further evidence that M1 requires L.

Sommer, S S, Wickner, R B

We describe two sets of plasmid-plasmid interactions in the yeast Saccharomyces cerevisiae. [HOK], [EXL], [NEX], and [KIL-k1] are genetically defined plasmids, and M1 and L are biochemically defined...

Genotypic detection of Mycobacterium tuberculosis rifampin resistance: comparison of single-strand conformation polymorphism and dideoxy fingerprinting.

Felmlee, T A, Liu, Q, Whelen, A C, Williams, D, Sommer, S S, Persing, D H

Detection of mutations in the rpoB gene of Mycobacterium tuberculosis can be used as an accurate predictor of rifampin resistance in the majority of strains tested. Simple but highly accurate...

Novel pattern of P53 mutation in breast cancers from Austrian women.

Hartmann, A, Rosanelli, G, Blaszyk, H, Cunningham, J M, McGovern, R M, Schroeder, J J, ...

Since mutagens produce an extraordinary diversity of mutational patterns, differential mutational exposures among populations are expected to produce different patterns of mutation. Classical...

Haemophilia B: database of point mutations and short additions and deletions, fifth edition, 1994.

Giannelli, F, Green, P M, Sommer, S S, Lillicrap, D P, Ludwig, M, Schwaab, R, ...

The fifth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of < 30bp)...

Screening for mutations by RNA single-strand conformation polymorphism (rSSCP): comparison with DNA-SSCP.

Sarkar, G, Yoon, H S, Sommer, S S

Single-strand conformation polymorphism (SSCP) is a simple method for detecting the presence of mutations in a segment of DNA, but the fraction of all mutations detected is unclear. We have evaluated...

Segments containing alternating purine and pyrimidine dinucleotides: patterns of polymorphism in humans and prevalence throughout phylogeny.

Sarkar, G, Paynton, C, Sommer, S S

Tandem dinucleotide repeats of GT or AC [(GT)n/(AC)n] where n greater than or equal to 14 are highly polymorphic and other simple repeats such as (CT)n/(AG)n and (A)n(T)n are also polymorphic. The...

The yeast KRE5 gene encodes a probable endoplasmic reticulum protein required for (1----6)-beta-D-glucan synthesis and normal cell growth.

Meaden, P, Hill, K, Wagner, J, Slipetz, D, Sommer, S S, Bussey, H

Yeast kre mutants define a pathway of cell wall (1----6)-beta-D-glucan synthesis, and mutants in genes KRE5 and KRE6 appear to interact early in such a pathway. We have cloned KRE5, and the sequence...

Superkiller mutations in Saccharomyces cerevisiae suppress exclusion of M2 double-stranded RNA by L-A-HN and confer cold sensitivity in the presence of M and L-A-HN.

Ridley, S P, Sommer, S S, Wickner, R B

In an mktl host, L-A-HN double-stranded RNA excludes M2 double-stranded RNA at 30 degrees C but not at 20 degrees C. Recessive mutations suppressing the exclusion of M2 by L-A-HN in an mktl host...

Double-stranded RNAs that encode killer toxins in Saccharomyces cerevisiae: unstable size of M double-stranded RNA and inhibition of M2 replication by M1.

Sommer, S S, Wickner, R B

The sizes of M1 and M2 (but not L) change rapidly with growth, varying by perhaps as much as 33%. Size variation is seen within 76 generations. In addition, the exclusion of M2 by M1 or L-A-E [(...

Germ-line origins of mutation in families with hemophilia B: the sex ratio varies with the type of mutation.

Ketterling, R P, Vielhaber, E, Bottema, C D, Schaid, D J, Cohen, M P, Sexauer, C L, ...

Previous epidemiological and biochemical studies have generated conflicting estimates of the sex ratio of mutation. Direct genomic sequencing in combination with haplotype analysis extends previous...

"Cryptic" repeating triplets of purines and pyrimidines (cRRY(i)) are frequent and polymorphic: analysis of coding cRRY(i) in the proopiomelanocortin (POMC) and TATA-binding protein (TBP) genes.

Gostout, B, Liu, Q, Sommer, S S

Triplets of the form of purine, purine, pyrimidine (RRY(i)) are enhanced in frequency in the genomes of primates, rodents, and bacteria. Some RRY(i) are "cryptic" repeats (cRRY(i)) in which no one...

Genotype relative risks: methods for design and analysis of candidate-gene association studies.

Schaid, D J, Sommer, S S

Design and analysis methods are presented for studying the association of a candidate gene with a disease by using parental data in place of nonrelated controls. This alternative design eliminates...

Characterization of the patterns of polymorphism in a "cryptic repeat" reveals a novel type of hypervariable sequence.

Jacobson, D P, Schmeling, P, Sommer, S S

Alternating purine and pyrimidine repeats (RY(i)) are an abundant source of polymorphism. The subset with long tandem repeats of GT or AC (GT(i)) have been studied extensively, but cryptic RY(i)...

From molecular variant to disease: initial steps in evaluating the association of transthyretin M119 with disease.

Ii, S, Sobell, J L, Sommer, S S

Traditionally, clinical research has sought to determine the molecular basis of clinical signs and symptoms. Increasingly, the traditional process will be reversed, as many structural protein...

Missense mutations and evolutionary conservation of amino acids: evidence that many of the amino acids in factor IX function as "spacer" elements.

Bottema, C D, Ketterling, R P, Ii, S, Yoon, H S, Phillips, J A, Sommer, S S

We report 31 point mutations in the factor IX gene and explore the relationship between the level of evolutionary conservation of an amino acid and the probability of a mutation causing hemophilia B....

Why does the human factor IX gene have a G + C content of 40%?

Bottema, C D, Bottema, M J, Ketterling, R P, Yoon, H S, Janco, R L, Phillips, J A, ...

The factor IX gene has a G + C content of approximately 40% in all mammalian species examined. In human factor IX, C----T and G----A transitions at the dinucleotide CpG are elevated at least 24-fold...

Functionally important regions of the factor IX gene have a low rate of polymorphism and a high rate of mutation in the dinucleotide CpG.

Koeberl, D D, Bottema, C D, Buerstedde, J M, Sommer, S S

We have recently described genomic amplification with transcript sequencing (GAWTS), a three-step procedure that allows direct genomic sequencing. By GAWTS more than 100,000 bp of sequence have been...

The pattern of factor IX germ-line mutation in Asians is similar to that of Caucasians.

Bottema, C D, Ketterling, R P, Yoon, H S, Sommer, S S

To begin documenting the pattern of germ-line mutations in different human races, we have delineated the mutation in nine Korean families with hemophilia B by direct genomic sequencing of the regions...

Mutations causing hemophilia B: direct estimate of the underlying rates of spontaneous germ-line transitions, transversions, and deletions in a human gene.

Koeberl, D D, Bottema, C D, Ketterling, R P, Bridge, P J, Lillicrap, D P, Sommer, S S

Spontaneous mutation provides the substrate for evolution on one hand and for genetic susceptibility to disease on the other hand. X-linked diseases such as hemophilia B offer an opportunity to...

Mutation detection by highly sensitive methods indicates that p53 gene mutations in breast cancer can have important prognostic value.

Kovach, J S, Hartmann, A, Blaszyk, H, Cunningham, J, Schaid, D, Sommer, S S

Human cancer cells with a mutated p53 tumor-suppressor gene have a selective growth advantage and may exhibit resistance to ionizing radiation and certain chemotherapeutic agents. To examine the...

Haemophilia B (sixth edition): a database of point mutations and short additions and deletions.

Giannelli, F, Green, P M, Sommer, S S, Poon, M C, Ludwig, M, Schwaab, R, ...

The sixth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of

Haemophilia B: database of point mutations and short additions and deletions, 7th edition.

Giannelli, F, Green, P M, Sommer, S S, Poon, M C, Ludwig, M, Schwaab, R, ...

The seventh edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of

Haemophilia B: database of point mutations and short additions and deletions--eighth edition.

Giannelli, F, Green, P M, Sommer, S S, Poon, M, Ludwig, M, Schwaab, R, ...

The eighth edition of the haemophilia B database (http://www.umds.ac. uk/molgen/haemBdatabase.htm ) lists in an easily accessible form all known factor IX mutations due to small changes (base...

Co-curing of plasmids affecting killer double-stranded RNAs of Saccharomyces cerevisiae: [HOK], [NEX], and the abundance of L are related and further evidence that M1 requires L.

Sommer, S S, Wickner, R B

We describe two sets of plasmid-plasmid interactions in the yeast Saccharomyces cerevisiae. [HOK], [EXL], [NEX], and [KIL-k1] are genetically defined plasmids, and M1 and L are biochemically defined...

Genotypic detection of Mycobacterium tuberculosis rifampin resistance: comparison of single-strand conformation polymorphism and dideoxy fingerprinting.

Felmlee, T A, Liu, Q, Whelen, A C, Williams, D, Sommer, S S, Persing, D H

Detection of mutations in the rpoB gene of Mycobacterium tuberculosis can be used as an accurate predictor of rifampin resistance in the majority of strains tested. Simple but highly accurate...

Novel pattern of P53 mutation in breast cancers from Austrian women.

Hartmann, A, Rosanelli, G, Blaszyk, H, Cunningham, J M, McGovern, R M, Schroeder, J J, ...

Since mutagens produce an extraordinary diversity of mutational patterns, differential mutational exposures among populations are expected to produce different patterns of mutation. Classical...

Haemophilia B: database of point mutations and short additions and deletions, fifth edition, 1994.

Giannelli, F, Green, P M, Sommer, S S, Lillicrap, D P, Ludwig, M, Schwaab, R, ...

The fifth edition of the haemophilia B database lists in easily accessible form all known factor IX mutations due to small changes (base substitutions and short additions and/or deletions of < 30bp)...

Screening for mutations by RNA single-strand conformation polymorphism (rSSCP): comparison with DNA-SSCP.

Sarkar, G, Yoon, H S, Sommer, S S

Single-strand conformation polymorphism (SSCP) is a simple method for detecting the presence of mutations in a segment of DNA, but the fraction of all mutations detected is unclear. We have evaluated...

Segments containing alternating purine and pyrimidine dinucleotides: patterns of polymorphism in humans and prevalence throughout phylogeny.

Sarkar, G, Paynton, C, Sommer, S S

Tandem dinucleotide repeats of GT or AC [(GT)n/(AC)n] where n greater than or equal to 14 are highly polymorphic and other simple repeats such as (CT)n/(AG)n and (A)n(T)n are also polymorphic. The...

The yeast KRE5 gene encodes a probable endoplasmic reticulum protein required for (1----6)-beta-D-glucan synthesis and normal cell growth.

Meaden, P, Hill, K, Wagner, J, Slipetz, D, Sommer, S S, Bussey, H

Yeast kre mutants define a pathway of cell wall (1----6)-beta-D-glucan synthesis, and mutants in genes KRE5 and KRE6 appear to interact early in such a pathway. We have cloned KRE5, and the sequence...

Superkiller mutations in Saccharomyces cerevisiae suppress exclusion of M2 double-stranded RNA by L-A-HN and confer cold sensitivity in the presence of M and L-A-HN.

Ridley, S P, Sommer, S S, Wickner, R B

In an mktl host, L-A-HN double-stranded RNA excludes M2 double-stranded RNA at 30 degrees C but not at 20 degrees C. Recessive mutations suppressing the exclusion of M2 by L-A-HN in an mktl host...

Double-stranded RNAs that encode killer toxins in Saccharomyces cerevisiae: unstable size of M double-stranded RNA and inhibition of M2 replication by M1.

Sommer, S S, Wickner, R B

The sizes of M1 and M2 (but not L) change rapidly with growth, varying by perhaps as much as 33%. Size variation is seen within 76 generations. In addition, the exclusion of M2 by M1 or L-A-E [(...

Germ-line origins of mutation in families with hemophilia B: the sex ratio varies with the type of mutation.

Ketterling, R P, Vielhaber, E, Bottema, C D, Schaid, D J, Cohen, M P, Sexauer, C L, ...

Previous epidemiological and biochemical studies have generated conflicting estimates of the sex ratio of mutation. Direct genomic sequencing in combination with haplotype analysis extends previous...

"Cryptic" repeating triplets of purines and pyrimidines (cRRY(i)) are frequent and polymorphic: analysis of coding cRRY(i) in the proopiomelanocortin (POMC) and TATA-binding protein (TBP) genes.

Gostout, B, Liu, Q, Sommer, S S

Triplets of the form of purine, purine, pyrimidine (RRY(i)) are enhanced in frequency in the genomes of primates, rodents, and bacteria. Some RRY(i) are "cryptic" repeats (cRRY(i)) in which no one...

Genotype relative risks: methods for design and analysis of candidate-gene association studies.

Schaid, D J, Sommer, S S

Design and analysis methods are presented for studying the association of a candidate gene with a disease by using parental data in place of nonrelated controls. This alternative design eliminates...

Characterization of the patterns of polymorphism in a "cryptic repeat" reveals a novel type of hypervariable sequence.

Jacobson, D P, Schmeling, P, Sommer, S S

Alternating purine and pyrimidine repeats (RY(i)) are an abundant source of polymorphism. The subset with long tandem repeats of GT or AC (GT(i)) have been studied extensively, but cryptic RY(i)...

From molecular variant to disease: initial steps in evaluating the association of transthyretin M119 with disease.

Ii, S, Sobell, J L, Sommer, S S

Traditionally, clinical research has sought to determine the molecular basis of clinical signs and symptoms. Increasingly, the traditional process will be reversed, as many structural protein...

Missense mutations and evolutionary conservation of amino acids: evidence that many of the amino acids in factor IX function as "spacer" elements.

Bottema, C D, Ketterling, R P, Ii, S, Yoon, H S, Phillips, J A, Sommer, S S

We report 31 point mutations in the factor IX gene and explore the relationship between the level of evolutionary conservation of an amino acid and the probability of a mutation causing hemophilia B....

Why does the human factor IX gene have a G + C content of 40%?

Bottema, C D, Bottema, M J, Ketterling, R P, Yoon, H S, Janco, R L, Phillips, J A, ...

The factor IX gene has a G + C content of approximately 40% in all mammalian species examined. In human factor IX, C----T and G----A transitions at the dinucleotide CpG are elevated at least 24-fold...

Functionally important regions of the factor IX gene have a low rate of polymorphism and a high rate of mutation in the dinucleotide CpG.

Koeberl, D D, Bottema, C D, Buerstedde, J M, Sommer, S S

We have recently described genomic amplification with transcript sequencing (GAWTS), a three-step procedure that allows direct genomic sequencing. By GAWTS more than 100,000 bp of sequence have been...

The pattern of factor IX germ-line mutation in Asians is similar to that of Caucasians.

Bottema, C D, Ketterling, R P, Yoon, H S, Sommer, S S

To begin documenting the pattern of germ-line mutations in different human races, we have delineated the mutation in nine Korean families with hemophilia B by direct genomic sequencing of the regions...

Mutations causing hemophilia B: direct estimate of the underlying rates of spontaneous germ-line transitions, transversions, and deletions in a human gene.

Koeberl, D D, Bottema, C D, Ketterling, R P, Bridge, P J, Lillicrap, D P, Sommer, S S

Spontaneous mutation provides the substrate for evolution on one hand and for genetic susceptibility to disease on the other hand. X-linked diseases such as hemophilia B offer an opportunity to...

The rates and patterns of deletions in the human factor IX gene.

Ketterling, R. P., Vielhaber, E. L., Lind, T. J., Thorland, E. C., Sommer, S. S.

Deletions are commonly observed in genes with either segments of highly homologous sequences or excessive gene length. However, in the factor IX gene and in most genes, deletions (of > or = 21 bp)...

The rates of G:C-->T:A and G:C-->C:G transversions at CpG dinucleotides in the human factor IX gene.

Ketterling, R. P., Vielhaber, E., Sommer, S. S.

We have identified eight independent transversions at CpG in 290 consecutive families with hemophilia B. These eight transversions account for 16.3% of all independent transversions in our sample,...

Comparison of statistics for candidate-gene association studies using cases and parents.

Schaid, D. J., Sommer, S. S.

Studies of association between candidate genes and disease can be designed to use cases with disease, and in place of nonrelated controls, their parents. The advantage of this design is the...

PAP-LMPCR for improved, allele-specific footprinting and automated chromatin fine structure analysis

Ingram, R., Gao, C., LeBon, J., Liu, Q., Mayoral, R. J., Sommer, S. S., ...

The analysis of chromatin fine structure and transcription factor occupancy of differentially expressed genes by in vivo footprinting and ligation-mediated-PCR (LMPCR) is a powerful tool to...

High frequency of p53 gene mutations in primary breast cancers in Japanese women, a low-incidence population.

Hartmann, A., Blaszyk, H., Saitoh, S., Tsushima, K., Tamura, Y., Cunningham, J. M., ...

The pattern of acquired mutations in the p53 tumour-suppressor gene is potentially useful for determining factors contributing to carcinogenesis in diverse populations differing in incidence and/or...