Epigenetic Telomere Protection by Drosophila DNA Damage Response Pathways: A Dissertation (2006)
Several aspects of Drosophila telomere biology indicate that telomere protection can be regulated by an epigenetic mechanism. First, terminally deleted chromosomes can be stably inherited and do not...
Epigenetic telomere protection by Drosophila DNA damage response pathways (2006)
Oikemus, Sarah R., Queiroz-Machado, Joana, Lai, KuanJu, McGinnis-Schultz, Nadine, Sunkel, Claudio E., Brodsky, Michael H.
Analysis of terminal deletion chromosomes indicates that a sequence-independent mechanism regulates protection of Drosophila telomeres. Mutations in Drosophila DNA damage response genes such as...
Epigenetic Telomere Protection by Drosophila DNA Damage Response Pathways (2006)
Sarah R. Oikemus, Joana Queiroz-Machado, KuanJu Lai, Nadine McGinnis, Claudio Sunkel, Michael H. Brodsky
Analysis of terminal deletion chromosomes indicates that a sequence-independent mechanism regulates protection of Drosophila telomeres. Mutations in Drosophila DNA damage response genes such as...
Epigenetic telomere protection by Drosophila DNA damage response pathways (2006)
Sarah R Oikemus, Joana Queiroz-Machado, KuanJu Lai, Nadine M McGinnis, Claudio E. Sunkel, Michael H Brodsky
Analysis of terminal deletion chromosomes indicates that a sequence-independent mechanism regulates protection of Drosophila telomeres. Mutations in Drosophila DNA damage response genes such as...
Epigenetic telomere protection by Drosophila DNA damage response pathways : a dissertation / (2006)
Thesis (Ph.D.)--University of Massachusetts Graduate School of Biomedical Sciences, 2006.
Oikemus, Sarah R., McGinnis-Schultz, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Castilla, Lucio H., Perrat, Paola N., Martinez, Natalia J., Landrette, Sean F., Keys, R., Oikemus, Sarah R., ...
Acute myeloid leukemia subtype M4 with eosinophilia is associated with a chromosome 16 inversion that creates a fusion gene CBFB-MYH11. We have previously shown that CBFB-MYH11 is necessary but not...
Oikemus, Sarah R., McGinnis, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Oikemus, Sarah R., McGinnis, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Oikemus, Sarah R., McGinnis, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Lee, Thomas H., Huang, Qiaojia, Oikemus, Sarah R., Shank, Jennifer, Ventura, Juan-Jose, Cusson, Nicole, ...
The cytokine tumor necrosis factor alpha (TNF-alpha) stimulates the NF-kappaB, SAPK/JNK, and p38 mitogen-activated protein (MAP) kinase pathways by recruiting RIP1 and TRAF2 proteins to the tumor...
Hermance, Nicole M., Oikemus, Sarah R., Kilpatrick, Elizabeth D., Cunningham, Leslie A., Kelliher, Michelle
Fas and the tumor necrosis factor receptor (TNFR)1 regulate the programmed cell death of lymphocytes. The death domain kinase, receptor interacting protein (rip), is recruited to the TNFR1 upon...
The DNA binding activity of TAL-1 is not required to induce leukemia/lymphoma in mice (2001)
O'Neil, Jennifer Elinor, Billa, Marilisa, Oikemus, Sarah R., Kelliher, Michelle
Activation of the basic helix-loop-helix (bHLH) gene TAL-1 (or SCL) is the most frequent gain-of-function mutation in pediatric T cell acute lymphoblastic leukemia (T-ALL). Similarly, mis-expression...
Oikemus, Sarah R., McGinnis, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Epigenetic Telomere Protection by Drosophila DNA Damage Response Pathways
Oikemus, Sarah R, Queiroz-Machado, Joana, Lai, KuanJu, McGinnis, Nadine, Sunkel, Claudio, Brodsky, Michael H
Analysis of terminal deletion chromosomes indicates that a sequence-independent mechanism regulates protection of Drosophila telomeres. Mutations in Drosophila DNA damage response genes such as...
Oikemus, Sarah R., McGinnis, Nadine, Queiroz-Machado, Joana, Tukachinsky, Hanna, Takada, Saeko, Sunkel, Claudio E., ...
Terminal deletions of Drosophila chromosomes can be stably protected from end-to-end fusion despite the absence of all telomere-associated sequences. The sequence-independent protection of these...
Epigenetic Telomere Protection by Drosophila DNA Damage Response Pathways
Oikemus, Sarah R, Queiroz-Machado, Joana, Lai, KuanJu, McGinnis, Nadine, Sunkel, Claudio, Brodsky, Michael H
Analysis of terminal deletion chromosomes indicates that a sequence-independent mechanism regulates protection of Drosophila telomeres. Mutations in Drosophila DNA damage response genes such as...