A highly flexible evaluation system for telecommunication analog front-ends (2001)
Bresch, M., Hammerschmidt, D., Hosticka, B.J., Kemmerling, M., Klein, B., Kokozinski, R., ...
As technological progress leads to increasing data transmission rates, use of digital transmission is inevitable. However, since analog waveforms are involved in digital transmission, analog...
Takahashi, K, Murayama, Y, Okamoto, T, Yokota, T, Ikezu, T, Takahashi, S, ...
The 14-residue peptide (peptide 14) corresponding to Arg2410-Lys2423 of the insulin-like growth factor II receptor (IGF-IIR) can activate the adenylate cyclase-inhibitor guanine nucleotide-binding...
Negative transactivation of cAMP response element by familial Alzheimer's mutants of APP.
Ikezu, T, Okamoto, T, Komatsuzaki, K, Matsui, T, Martyn, J A, Nishimoto, I
In familial Alzheimer's disease (FAD), missense point mutations V642I/F/G, which co-segregate with the disease phenotype, have been discovered in amyloid precursor APP695. Here, we report that three...
G protein betagamma complex-mediated apoptosis by familial Alzheimer's disease mutant of APP.
Giambarella, U, Yamatsuji, T, Okamoto, T, Matsui, T, Ikezu, T, Murayama, Y, ...
In familial Alzheimer's disease (FAD), three missense mutations, V642I, V642F and V642G, that co-segregate with the disease phenotype have been discovered in the 695 amino acid form of the amyloid...
Takahashi, K, Murayama, Y, Okamoto, T, Yokota, T, Ikezu, T, Takahashi, S, ...
The 14-residue peptide (peptide 14) corresponding to Arg2410-Lys2423 of the insulin-like growth factor II receptor (IGF-IIR) can activate the adenylate cyclase-inhibitor guanine nucleotide-binding...
Negative transactivation of cAMP response element by familial Alzheimer's mutants of APP.
Ikezu, T, Okamoto, T, Komatsuzaki, K, Matsui, T, Martyn, J A, Nishimoto, I
In familial Alzheimer's disease (FAD), missense point mutations V642I/F/G, which co-segregate with the disease phenotype, have been discovered in amyloid precursor APP695. Here, we report that three...
G protein betagamma complex-mediated apoptosis by familial Alzheimer's disease mutant of APP.
Giambarella, U, Yamatsuji, T, Okamoto, T, Matsui, T, Ikezu, T, Murayama, Y, ...
In familial Alzheimer's disease (FAD), three missense mutations, V642I, V642F and V642G, that co-segregate with the disease phenotype have been discovered in the 695 amino acid form of the amyloid...