Thomas Eissing

Bistability preserving model reduction in apoptosis (2007)

Waldherr, Steffen, Eissing, Thomas, Chaves, Madalena, Allgower, Frank

Biological systems are typically very complex and need to be reduced before they are amenable to a thorough analysis. Also, they often possess functionally important dynamic features like...

Response to bistability in apoptosis: Roles of Bax, Bcl-2, and mitochondrial permeability transition pores (2007)

Eissing, Thomas, Waldherr, Steffen, Allgöwer, Frank, Scheurich, Peter, Bullinger, Eric

Recently, a mathematical model of the mitochondrial apoptotic pathway was proposed. In that study, the robustness of different simplified signaling models with respect to parameter changes was also...

Steady state and (bi-) stability evaluation of simple protease signalling networks (2007)

Eissing, Thomas, Waldherr, Steffen, Allgöwer, Frank, Scheurich, Peter, Bullinger, Eric

Signal transduction networks are complex, as are their mathematical models. Gaining a deeper understanding requires a system analysis. Important aspects are the number, location and stability of...

Bistability analyses of a caspase activation model for receptor-induced apoptosis (2004)

Eissing, Thomas, Conzelmann, Holger, Gilles, Ernst Dieter, Allgöwer, Frank, Bullinger, Eric, Scheurich, Peter

Apoptosis is an important physiological process crucially involved in development and homeostasis of multicellular organisms. Although the major signaling pathways have been unraveled, a detailed...