Tim Wiltshire

Expression analysis of G Protein-Coupled Receptors in mouse macrophages (2008)

Lattin, Jane E, Schroder, Kate, Su, Andrew I, Walker, John R, Zhang, Jie, Wiltshire, Tim, ...

Abstract Background Monocytes and macrophages express an extensive repertoire of G Protein-Coupled Receptors (GPCRs) that regulate inflammation and immunity. In this study we performed a systematic...

Coordinated diurnal regulation of genes from the Dlk1 Dio3 imprinted domain: implications for regulation of clusters of non-paralogous genes (2008)

Labialle, Stéphane, Yang, Lanjian, Ruan, Xuan, Villemain, Aude, Schmidt, Jennifer V., Hernandez, Arturo, ...

The functioning of the genome is tightly related to its architecture. Therefore, understanding the relationship between different regulatory mechanisms and the organization of chromosomal domains is...

Utilization of a whole genome SNP panel for efficient genetic mapping in the mouse (2006)

Moran, Jennifer L., Bolton, Andrew D., Tran, Pamela V., Brown, Alison, Dwyer, Noelle D., Manning, Danielle K., ...

Phenotype-driven genetics can be used to create mouse models of human disease and birth defects. However, the utility of these mutant models is limited without identification of the causal gene. To...

A mutant mouse with a highly specific contextual fear-conditioning deficit found in an N-ethyl-N-nitrosourea (ENU) mutagenesis screen (2006)

Reijmers, Leon G., Coats, Jennifer K., Pletcher, Mathew T., Wiltshire, Tim, Tarantino, Lisa M., Mayford, Mark

Targeted mutagenesis in mice has shown that genes from a wide variety of gene families are involved in memory formation. The efficient identification of genes involved in learning and memory could be...

Utilization of a whole genome SNP panel for efficient genetic mapping in the mouse (2006)

Moran, Jennifer L., Bolton, Andrew D., Tran, Pamela V., Brown, Alison, Dwyer, Noelle D., Manning, Danielle K., ...

Phenotype-driven genetics can be used to create mouse models of human disease and birth defects. However, the utility of these mutant models is limited without identification of the causal gene. To...

Uncovering regulatory pathways that effect hematopoietic stem cell function using 'genetical genomics' (2005)

Bystrykh, Leonid, Weersing, Ellen, Dontje, Bert, Sutton, Sue, Pletcher, Mathew T., Wiltshire, Tim, ...

We combined large-scale mRNA expression analysis and gene mapping to identify genes and loci that control hematopoietic stem cell (HSC) function. We measured mRNA expression levels in purified HSCs...

BMC Bioinformatics BioMed Central Research article Comparative analysis of haplotype association mapping algorithms (2005)

Phillip Mcclurg, Mathew T Pletcher, Tim Wiltshire, Andrew I Su, Andrew I Su

© 2006McClurg et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License

Genome-wide linkage identifies novel modifier loci of aganglionosis in the Sox10Dom model of Hirschsprung disease (2005)

Owens, Sarah E., Broman, Karl W., Wiltshire, Tim, Elmore, J. Bradford, Bradley, Kevin M., Smith, Jeffrey R., ...

Hirschsprung disease (HSCR) is a complex disorder that exhibits incomplete penetrance and variable expressivity due to interactions among multiple susceptibility genes. Studies in HSCR families have...

Genome-wide Linkage Identifies Novel Modifier Loci of Aganglionosis in the Sox10Dom Model of Hirschsprung Disease (2005)

Owens, Sarah E., Broman, Karl W., Wiltshire, Tim, Elmore, J. Bradford, Bradley, Kevin M., Smith, Jeffrey R., ...

Hirschsprung disease (HSCR) is a complex disorder that exhibits incomplete penetrance and variable expressivity due to interactions among multiple susceptibility genes. Studies in HSCR families have...

Use of a Dense Single Nucleotide Polymorphism Map for In Silico Mapping in the Mouse (2004)

Mathew T. Pletcher, Philip McClurg, Serge Batalov, Andrew I. Su, S. Whitney Barnes, Erica Lagler, ...

Rapid expansion of available data, both phenotypic and genotypic, for multiple strains of mice has enabled the development of new methods to interrogate the mouse genome for functional genetic...

Gene Set Enrichment in eQTL Data Identifies Novel Annotations and Pathway Regulators

Wu, Chunlei, Delano, David L., Mitro, Nico, Su, Stephen V., Janes, Jeff, McClurg, Phillip, ...

Genome-wide gene expression profiling has been extensively used to generate biological hypotheses based on differential expression. Recently, many studies have used microarrays to measure gene...

The Modifier of hemostasis (Mh) locus on chromosome 4 controls in vivo hemostasis of Gp6−/− mice

Cheli, Yann, Jensen, Deborah, Marchese, Patrizia, Habart, David, Wiltshire, Tim, Cooke, Michael, ...

Platelet glycoprotein VI (GPVI) is a key receptor for collagens that mediates the propagation of platelet attachment and activation. Targeted disruption of the murine gene Gp6 on a mixed 129 × 1/SvJ...

A Common and Unstable Copy Number Variant Is Associated with Differences in Glo1 Expression and Anxiety-Like Behavior

Williams, Richard, Lim, Jackie E., Harr, Bettina, Wing, Claudia, Walters, Ryan, Distler, Margaret G., ...

Glyoxalase 1 (Glo1) has been implicated in anxiety-like behavior in mice and in multiple psychiatric diseases in humans. We used mouse Affymetrix exon arrays to detect copy number variants (CNV)...

The genetic contribution to heart rate and heart rate variability in quiescent mice

Howden, Reuben, Liu, Eric, Miller-DeGraff, Laura, Keener, Heather L., Walker, Christopher, Clark, James A., ...

Recent studies have suggested a genetic component to heart rate (HR) and HR variability (HRV). However, a systematic examination of the genetic contribution to the variation in HR and HRV has not...

A mouse model for nonsyndromic deafness (DFNB12) links hearing loss to defects in tip links of mechanosensory hair cells

Schwander, Martin, Xiong, Wei, Tokita, Joshua, Lelli, Andrea, Elledge, Heather M., Kazmierczak, Piotr, ...

Deafness is the most common form of sensory impairment in humans and is frequently caused by single gene mutations. Interestingly, different mutations in a gene can cause syndromic and nonsyndromic...