Vesa Kaartinen

Publication List Details

Period

1991 - 2009

Number

23

Co-Authors

The type I BMP receptors, Bmpr1a and Acvr1, activate multiple signaling pathways to regulate lens formation (2009)

Huang, Jie, Dattilo, Lisa K, Kaartinen, Vesa, Mishina, Yuji, Deng, Chu-Xia, ...

BMPs play multiple roles in development and BMP signaling is essential for lens formation. However, the mechanisms by which BMP receptors function in vertebrate development are incompletely...

FGF-regulated BMP signaling is required for eyelid closure and to specify conjunctival epithelial cell fate (2009)

Dattilo, Lisa K, Rajagopal, Ramya, Liu, Ying, Kaartinen, Vesa, Mishina, Yuji, ...

There are conflicting reports about whether BMP signaling is required for eyelid closure during fetal development. This question was addressed using mice deficient in BMP or TGF beta signaling in...

Signaling via Alk5 Controls Ontogeny of Lung Clara Cells (2009)

Yiming Xing, Changgong Li, Aimin Li, Somyoth Sridurongrit, Caterina Tiozzo, Saverio Bellusci, ...

Clara cells, together with ciliated and pulmonary neuroendocrine cells, make up the epithelium of the bronchioles along the conducting airways. Clara cells are also known as Progenitor/Stem cells...

Defective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defects (2006)

Wang, Jikui, Nagy, Andre, Larsson, Jonas, Dudas, Marek, Sucov, Henry M, Kaartinen, Vesa

Abstract Background Congenital cardiovascular diseases are the most common form of birth defects in humans. A substantial portion of these defects has been associated with inappropriate induction,...

BMC Developmental Biology BioMed Central (2006)

Jikui Wang, Andre Nagy, Jonas Larsson, Marek Dudas, Henry M Sucov, Vesa Kaartinen

Research article Defective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defects

Growth factor signaling in lung morphogenetic centers: automaticity, stereotypy and symmetry (2003)

Warburton, David, Bellusci, Saverio, Del Moral, Pierre-Marie, Kaartinen, Vesa, Lee, Matt, Tefft, Denise, ...

Abstract Lung morphogenesis is stereotypic, both for lobation and for the first several generations of airways, implying mechanistic control by a well conserved, genetically hardwired developmental...

Respiratory Research BioMed Central Review (2003)

David Warburton, Saverio Bellusci, Vesa Kaartinen, Matt Lee, Denise Tefft, ...

Growth factor signaling in lung morphogenetic centers: automaticity, stereotypy and symmetry

Glycosaparaginase from human leukocytes. Inactivation and covalent modification with diazo-oxonorvaline (1991)

Kaartinen, Vesa, Williams, Julian C., Tomich, John, Hood, Leroy E., Mononen, Ilkka

The apparent active site of human leukocyte glycoasparaginase (N4-(beta-acetylglucosaminyl)-L-asparaginase EC 3.5.1.26) has been studied by labeling with an asparagine analogue,...

TGF-β3-induced Palatogenesis Requires Matrix Metalloproteinases

Blavier, Laurence, Lazaryev, Alisa, Groffen, John, Heisterkamp, Nora, DeClerck, Yves A., Kaartinen, Vesa

Cleft lip and palate syndromes are among the most common congenital malformations in humans. Mammalian palatogenesis is a complex process involving highly regulated interactions between epithelial...

Growth factor signaling in lung morphogenetic centers: automaticity, stereotypy and symmetry

Warburton, David, Bellusci, Saverio, Del Moral, Pierre-Marie, Kaartinen, Vesa, Lee, Matt, Tefft, Denise, ...

Lung morphogenesis is stereotypic, both for lobation and for the first several generations of airways, implying mechanistic control by a well conserved, genetically hardwired developmental program....

Generation of rac3 Null Mutant Mice: Role of Rac3 in Bcr/Abl-Caused Lymphoblastic Leukemia

Cho, Young Jin, Zhang, Bin, Kaartinen, Vesa, Haataja, Leena, De Curtis, Ivan, Groffen, John, ...

Numerous studies indirectly implicate Rac GTPases in cancer. To investigate if Rac3 contributes to normal or malignant cell function, we generated rac3 null mutants through gene targeting. These mice...

Atrioventricular cushion transformation is mediated by ALK2 in the developing mouse heart

Wang, Jikui, Sridurongrit, Somyoth, Dudas, Marek, Thomas, Penny, Nagy, Andre, Schneider, Michael D., ...

Developmental abnormalities in endocardial cushions frequently contribute to congenital heart malformations including septal and valvular defects. While compelling evidence has been presented to...

TGF-β3-induced Palatogenesis Requires Matrix Metalloproteinases

Blavier, Laurence, Lazaryev, Alisa, Groffen, John, Heisterkamp, Nora, DeClerck, Yves A., Kaartinen, Vesa

Cleft lip and palate syndromes are among the most common congenital malformations in humans. Mammalian palatogenesis is a complex process involving highly regulated interactions between epithelial...

Growth factor signaling in lung morphogenetic centers: automaticity, stereotypy and symmetry

Warburton, David, Bellusci, Saverio, Del Moral, Pierre-Marie, Kaartinen, Vesa, Lee, Matt, Tefft, Denise, ...

Lung morphogenesis is stereotypic, both for lobation and for the first several generations of airways, implying mechanistic control by a well conserved, genetically hardwired developmental program....

Generation of rac3 Null Mutant Mice: Role of Rac3 in Bcr/Abl-Caused Lymphoblastic Leukemia

Cho, Young Jin, Zhang, Bin, Kaartinen, Vesa, Haataja, Leena, De Curtis, Ivan, Groffen, John, ...

Numerous studies indirectly implicate Rac GTPases in cancer. To investigate if Rac3 contributes to normal or malignant cell function, we generated rac3 null mutants through gene targeting. These mice...

Abr and Bcr, Two Homologous Rac GTPase-Activating Proteins, Control Multiple Cellular Functions of Murine Macrophages▿ †

Cho, Young Jin, Cunnick, Jess M., Yi, Sun-Ju, Kaartinen, Vesa, Groffen, John, Heisterkamp, Nora

Small GTPases of the Rho family are key regulators of phagocytic leukocyte function. Abr and Bcr are homologous, multidomain proteins. Their C-terminal domain has GTPase-activating protein (GAP)...

Progressive Neurodegeneration in Aspartylglycosaminuria Mice

Gonzalez-Gomez, Ignacio, Mononen, Ilkka, Heisterkamp, Nora, Groffen, John, Kaartinen, Vesa

Aspartylglycosaminuria (AGU) is one of the most common lysosomal storage disorders in humans. A mouse model for AGU has been recently generated through targeted disruption of the glycosylasparaginase...

ALK5- and TGFBR2-independent role of ALK1 in the pathogenesis of hereditary hemorrhagic telangiectasia type 2

Park, Sung O., Lee, Young Jae, Seki, Tsugio, Hong, Kwon-Ho, Fliess, Naime, Jiang, Zhigang, ...

ALK1 belongs to the type I receptor family for transforming growth factor-β family ligands. Heterozygous ALK1 mutations cause hereditary hemorrhagic telangiectasia type 2 (HHT2), a multisystemic...